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The old ECG has a Q-wave with persistent ST elevation in lead III, and some reciprocal ST depression (typical for aneurysm morphology). This is "Persistent ST elevation after previous MI" or "LV aneurysm morphology". LV aneurysm is very different for inferior vs. anterior MI. The patient had a critical LAD stenosis.
An echocardiogram showed severely reduced global systolic function with an EF of 20-25% and an LV apical thrombus. The red arrow shows a roughly 80% stenosis of the proximal LAD. The blue arrow shows another stenosis of the LAD distal to the first diagonal branch of about 99%. The LV aneurysm morphology persists.
The EKG is diagnostic of acute inferior, posterior, and lateral OMI superimposed on “LV aneurysm” morphology. Additionally, a bedside echocardiogram showed no wall motion abnormality and normal LV function. Angiography revealed a 30% nonobstructive stenosis of the mid LAD. Patient 2 , EKG 1: What do you think?
hours after that first diagnostic ECG) : Mid-LAD culprit lesion, 99% stenosis, no pre-intervention TIMI flow available, but described as "severe subtotal lesion", which was stented with reported TIMI 3 flow resulting. Another lesion in the proximal LAD with 80% stenosis was stented as well. Culprit is 100% stenosis in the Proximal LAD.
Echocardiogram in parasternal long axis view shows dilated left ventricle, left atrium, aorta and a small portion of the right ventricle, which is usually the outflow region. When there is associated mitral stenosis, the colour Doppler jet of mitral flow merges with that of aortic regurgitation in the left ventricle as both occur in diastole.
Here are the images from the cardiac cath: Mid to distal-LAD in-stent stenosis with 100% occlusion and TIMI flow 0 LAD post-DES placement with TIMI 3 flow The amount of territory supplied by this vessel becomes obvious here (and goes on for a few more frames below this still). No further echocardiograms were available after cath.
Look for Vascular Etiology -- think of these while doing H and P: --Bleeding: ruptured AAA, GI bleed, ruptured ectopic pregnancy, other spontaneous bleed such as mesenteric aneurysms. Aortic Dissection, Valvular (especially Aortic Stenosis), Tamponade. heart auscultation (aortic stenosis); c. orthostatic vitals b.
There are no Q-waves to suggest old inferior MI, or inferior aneurysm as the etiology of the ST Elevation. Smith : "decompensation" of aortic stenosis might have initiated this entire cascade. What "initiates" the aortic stenosis cascade? The scan showed a bicuspid aortic valve with severe stenosis and coronary artery disease.
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