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Of course, papillary muscle rupture and mitral regurgitation should be on the differential here, as in this case , but it is not very likely when the BP is so high. Inferior LV "aneurysm" morphology Electrocardiographic "LV Aneurysm" morphology simply means "persistent ST elevation after previous MI." Is this acute STEMI?
Echocardiogram in parasternal long axis view shows dilated left ventricle, left atrium, aorta and a small portion of the right ventricle, which is usually the outflow region. Mitral valve leaflets seen in open position between the left ventricle and left atrium are thickened.
The post ectopic increase in the murmur is a hallmark of hypertrophic obstructive cardiomyopathy, which differentiates it clinically from mitral valve prolapse. Echocardiography in HCM Important echocardiographic features include mitral regurgitation and left ventricular outflow tract obstruction.
Next day echocardiogram showed inferolateral hypokinesia with an EF of %45-50. On echocardiogram you will not see a "posterior" hypokinesia (will see "inferolateral") and, as in this case, LCx may not give the blood supply of basal inferior segment (formerly called "posterior"). The patient recovered well.
Kawasaki Disease Case : Angela Khidhir, DO, MSc (USA) presented a compelling case on ACS and tamponade potentially linked to Kawasaki disease-related Coronary Artery Aneurysm. This debate underscored the growing role of CSP despite the current limited evidence supporting it.
There are no Q-waves to suggest old inferior MI, or inferior aneurysm as the etiology of the ST Elevation. The scan showed a bicuspid aortic valve with severe stenosis and coronary artery disease. However, there is also significant tachycardia , with heart rate of 116, and known hypoxia.
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