Remove Anatomy Remove Ischemia Remove Stenosis
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90 year old with acute chest and epigastric pain, and diffuse ST depression with reciprocal STE in aVR: activate the cath lab?

Dr. Smith's ECG Blog

The best course is to wait until the anatomy is defined by angio, then if proceeding to PCI, add Cangrelor (an IV P2Y12 inhibitor) I sent the ECG and clinical information of a 90-year old with chest pain to Dr. McLaren. His response: “subendocardial ischemia. A emergent cardiology consult can be helpful for equivocal cases.

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Inferior Subtle ST elevation: straight ST segment, but also no reciprocal ST depression in aVL: which is more important?

Dr. Smith's ECG Blog

The cath lab was activated: Result: Thrombotic 95% stenosis at the ostium of a small LPL2 with 70% stenosis at the LPL2/LPDA bifurcation in the distal/AV groove Cx Tubular 70% stenosis in the mid-circumflex. (In More likely, these T waves probably reflect ischemia of uncertain age. It was stented. Learning Points: 1.

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Chest pain, and Cardiology didn't take the hint from the ICD

Dr. Smith's ECG Blog

Ischemic ST-Segment Depression Maximal in V1-V4 (Versus V5-V6) of Any Amplitude Is Specific for Occlusion Myocardial Infarction (Versus Nonocclusive Ischemia). 90% stenosis of the proximal ramus intermedius, pre procedure TIMI II flow The ramus intermedius is a normal variant on coronary anatomy that arises between the LAD and LCX.

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Dueling OMI: does this 30 year old with chest pain have any signs of occlusion or reperfusion?

Dr. Smith's ECG Blog

For example, inferior OMI with concomitant critical stenosis produces a combined pattern ( Aslanger’s pattern ) with inferior STE and subendocardial ischemia · occlusion of two infarct-related arteries simultaneously ("co-culprits") In this case there were two infarct-related arteries.

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3 days of shoulder and chest pain, and now cardiogenic shock

Dr. Smith's ECG Blog

This is ischemia until proven otherwise. Here I annotate it: This shows 100% occluded circumflex (red arrow) and a 90% stenosis of the LAD (Yellow arrow). The LAD was thought to be not thrombotic, but a chronic tight stenosis. I learned that the patient is on Sotalol for control of PVCs. This explains the long QT.

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A "normal ECG" on a busy night

Dr. Smith's ECG Blog

If the patient had been "lucky," his symptoms from the prior day might have been due to ischemia prolonged and intense enough to result in small troponin increase. The anatomy and lead placement create very small voltage compared to the other main coronary distributions. EF was 55%. The patient did well at least in the short term.