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The Guidezilla guide extension catheter (GEC) with a larger diameter and extended length is widely used in challenging coronary anatomy. However, the constrained accessibility of dedicated catheters has impeded the potential benefits of standard CDT in developing countries.
a powerful and advanced mechanical thrombectomy system for the removal of venous thrombus and the treatment of pulmonary embolism (PE), effectively completing Penumbras VTE platform. The Element Vascular Access System is compatible with Lightning Flash 2.0, The flexibility and torqueability of the Lightning Flash2.0
The pivotal trial will study the Vertex Pulmonary Embolectomy System, which incorporates Jupiter’s Endoportal Control platform technology into an endovascular procedure intended to treat Acute Pulmonary Embolism (PE) with an unprecedented level of control and precision.
Left atrial appendage (LAA) occlusion is a promising strategy for reducing embolic events in atrial fibrillation. Additionally, the device integrates independent adaptive anchors that autonomously enhance stability & adaptability, accommodating diverse LAA anatomies without compromising device stability.
Blood Clots: An enlarged heart is more prone to developing blood clots, which can lead to stroke or pulmonary embolism. Cardiac Arrest or Sudden Death: Cardiomegaly increases the risk of life-threatening arrhythmias, which can cause sudden cardiac arrest.
Procedure related complications included: phrenic nerve palsy [temporary 4 (1.2%), persistent 0 (0.0%)], cardiac tamponade/perforation 2 (0.6%), and air embolism 1 (0.3%). Conclusions This novel cryoballoon may facilitate PVI to treat PAF, providing more options to address the variety of anatomies present in patients with PAF.
Mid cavity obstruction in HCM is associated with apical aneurysm, systemic embolism, and arrhythmias. Evaluation of diastolic characteristics of LV and LV and coronary anatomy evaluation are other diagnostic uses of cath in HCM. LVOTO is due to septal hypertrophy, SAM, and anterior displacement of mitral valve apparatus.
CT angiogram chest: no aortic dissection or pulmonary embolism. He spent several days in the PICU, undergoing workup including: Serial troponins: rising from 5,700 ng/L (unknown if I or T) to greater than 25,000 ng/L (greater than the lab's upper limit of reporting). No further troponins were measured.
Angiogram: --"Suspected culprit for the patient's non-ST elevation myocardial infarction with refractory chest discomfort (although it had resolved prior to arrival to the cardiac catheterization lab), is a ruptured plaque in the distal circumflex with local embolic occlusion of the distal OM 3."
His initial high sensitivity troponin I returned at 1300 ng/L and given that his cardiac workup was otherwise unremarkable, a CT was obtained to evaluate for pulmonary embolism and aortic aneurysm or dissection but this too was unrevealing. Another EKG was also obtained. ECG at time 82 minutes: What do you think?
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