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My written interpretation on a tracing such as this one would read, "Marked LVH and 'strain' and/or ischemia — with need for clinical correlation." BOTTOM LINE: ECG changes of LV "strain" and/or ischemia that we see on today's initial ECG — were not present 9 years earlier. WPW Cardiac arrhythmias ( including AFib ).
This is a very typical ECG for Hypertrophic Cardiomyopathy. The most recent previous was 4 years prior, and was in the normal range) Elderly patients, and patients with cardiomyopathy (including HOCM), may have troponin values in this range chronically ("chronic myocardial injury"). WPW Cardiac arrhythmias ( including AFib ).
(Harvard University Heart Letter) A clinical polygenic risk score test for diseases ranging from atrial fibrillation (AFib) to breast cancer was piloted by scientists. Patients with obstructive hypertrophic cardiomyopathy who underwent surgical myectomy reported improved quality of life.
Are you confident there is no ischemia? Primary VT , and the VT with tachycardia is causing ischemia with chest discomfort (supply-demand mismatch/type 2 MI)? Ischemia from ACS causing the chest discomfort, with VT another consequence (or coincidence)? Do you agree with this strategy? How can you better assess the ST segments?
The QRS is wide in B — but the rhythm is irregularly irregular with no sinus P waves — so this most probably represents rapid AFib with an atypical RBBB/LPHB morphology. We now see that QRS morphology in lead II during sinus rhythm is similar to the QRS morphology in lead II during rapid AFib (beats #1-5 in lead II in A).
Here was my answer: "Not ischemia. WPW Cardiac arrhythmias ( especially AFib ). If not HCM — some unusual form of cardiomyopathy might explain the findings in today's ECG (ie, muscular dystrophy; infiltrative heart disease from amyloid or sarcoid; some unusual form of congenital heart disease, etc. ). What do you think?
There is no evidence of infarction or ischemia. The absence of any wall motion abnormality makes ischemic cardiomyopathy very unlikely. The new onset cardiomyopathy was thought to be due to both drug/alcohol use and to Tachycardia-Induced Cardiomyopathy. There are nonspecific ST-T abnormalities.
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