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PMH: Known paroxysmal Atrial fib. He is usually is in sinus rhythm as far as he knows, but he cannot subjectively feel atrialfibrillation, so he is never completely certain when he is in sinus or atrial fib. Here is his ECG: Atrial Fib with a Ventricular Response of about 66. He immediately completely recovered.
Her Apple Watch suddenly told her that she is in atrialfibrillation. Patients with healthy AV nodes who are not on AV nodal blockers and who are not hyperkalemic should have a rapid ventricular response if they have paroxysmal Atrialfibrillation. I focus my comment on a few additional aspects regarding new AFib.
Written by Pendell Meyers First try to interpret this ECG with no clinical context: The ECG shows an irregularly irregular rhythm, therefore almost certainly atrialfibrillation. After an initially narrow QRS, there is a very large abnormal extra wave at the end of the QRS complex. There is also large T wave inversion and long QT.
Chart review confirmed that he had been started on flecainide for atrialfibrillation. This new information makes the diagnosis of atrial flutter far more likely: first, atrialfibrillation and flutter are closely associated and, second, this makes a flutter rate of 200 bpm (with 1:1 conduction) quite likely.
Atrial fib may cause Occlusion mimic." ACUTE MI (I allowed Acute MI to be in the report because I knew there would be an elevated troponin from ischemia, which is the definition of acute MI -- but in this case it would most likely be a Type 2 MI from tachycardia) There is also LA-RA lead reversal. The rhythm is rapid AFib.
She previously had Atrialfibrillation with LBBB. This shows atrialfibrillation. The fact that the response is regular proves that the atrialfibrillation is NOT conducting. When atrial fib conducts, the ventricular rate must always be irregular. There was no evidence of ischemia.
(Harvard University Heart Letter) A clinical polygenic risk score test for diseases ranging from atrialfibrillation (AFib) to breast cancer was piloted by scientists.
The medical care providers ascribed the patient's chest pain to new onset atrialfibrillation with rapid ventricular response after having viewed the ECG. The presentation ECG does show atrialfibrillation. No further episodes of atrialfibrillation occurred during monitoring. The first ECG is shown below.
The ECG shows sinus tachycardia with RBBB and LAFB, without clear additional superimposed signs of ischemia. Atrialfibrillation is also a predictor of worse outcomes in this case (Alborzi). Other Arrhythmias ( PACs, PVCs, AFib, Bradycardia and AV conduction disorders — potentially lethal VT/VFib ).
She also has a hx of paroxysmal atrialfibrillation and is on oral anticoagulant treatment. Are you confident there is no ischemia? The heart rate could be compatible with that of a 2:1 conducted atrial flutter. Ischemia from ACS causing the chest discomfort, with VT another consequence (or coincidence)?
The rhythm is atrialfibrillation. In terms of ischemia, there is both a signal of subendocardial ischemia (STD max in V5-V6 with reciprocal STE in aVR) AND a signal of transmural infarction of the inferior wall with Q wave and STE in lead III with reciprocal STD in I and aVL. The QRS complex is within normal limits.
Case submitted and written by Mazen El-Baba MD, with edits from Jesse McLaren and edits/comments by Smith and Grauer A 90-year old with a past medical history of atrialfibrillation, type-2 diabetes, hypertension, dyslipidemia, presented with acute onset chest/epigastric pain, nausea, and vomiting. Anything more on history?
Here is her post-cardioversion ECG: ECG#2 - Immediately post cardioversion: Appropriate ST depression maximal in V5-6 and lead II, secondary to subendocardial ischemia, likely residual from the preceding tachycardia. Patient was referred to electrophysiologic testing due to suspicion of afib and WPW. She was sedated and cardioverted.
There is a large peaked P-wave in lead II (right atrial enlargement) There is left axis deviation consistent with left anterior fascicular block. There is no evidence of infarction or ischemia. There are nonspecific ST-T abnormalities. Troponin I was 0.054 ng/mL NT-ProBNP was 8316 (0-900 pg/mL). "
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