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In this weeks View, Dr. Eagle looks at arrhythmia recurrence and rhythm control after catheter ablation for atrial fibrillation. He then discusses long-term outcomes in transthyretin amyloid cardiomyopathy in patients treated with tafamidis.
In this week’s View, Dr. Eagle looks at anticoagulation for post-operative atrial fibrillation after isolated coronary artery bypass grafting. He then examines a meta-analysis of invasive vs. conservative management of non-ST-elevation acute coronary syndromes with previous coronary artery bypass grafting.
Objective To report the number of thromboembolic complications in a cohort of pediatric and young adult patients presenting with atrial fibrillation (AFib) or atrial flutter (AFl) while also assessing anticoagulation practice in a multicenter cohort of young patients with these arrhythmias.
This is a very typical ECG for Hypertrophic Cardiomyopathy. The most recent previous was 4 years prior, and was in the normal range) Elderly patients, and patients with cardiomyopathy (including HOCM), may have troponin values in this range chronically ("chronic myocardial injury"). WPW Cardiac arrhythmias ( including AFib ).
That said — distinction between "classic" HCM vs the apical HCM for m may be useful because: i ) ECG findings tend to be different ( Lyon et al — Europace 20:102-112iii, 2018 ) ; — ii ) Echo appearance is different when hypertrophy localizes to the apex; and , iii ) There is a significantly greater incidence of AFib with apical HCM.
The patient was diagnosed with stress cardiomyopathy. Widespread T wave inversions and prolongation of the QT interval is not uncommon in Takotsubo cardiomyopathy. The QTc then gradually shortened over the course of several days as is usual for stress cardiomyopathy. Potassium was 4,8 mmol/l. ( ref 3,5-4,6 mmol/l ).
As a result, in order to differentiate MAT from the much more commonly encountered irregularly irregular rhythm ( which is AFib ) — we need to be certain we are seeing multiple different P wave morphologies that are constantly changing. Clinically , the importance of recognizing MAT — is that treatment is different than the treatment of AFib.
The QRS is wide in B — but the rhythm is irregularly irregular with no sinus P waves — so this most probably represents rapid AFib with an atypical RBBB/LPHB morphology. We now see that QRS morphology in lead II during sinus rhythm is similar to the QRS morphology in lead II during rapid AFib (beats #1-5 in lead II in A).
The patient also has a history of AFib and HFmrEF ( = H eart F ailure with M inimally- R educed E jection F raction ). There is almost always the possibility of exceptions ( ie, prior infarction or cardiomyopathy that might result in a very abnormal baseline tracing ). Why was it Wrong to Think the Rhythm was AFlutter?
He had a h/o ischemic cardiomyopathy and right MCA stroke. As per Dr. Smith — this suggests that despite QRS widening, the rhythm in ECG #3 is AFib with a rapid ventricular response. Given that the patient was asymptomatic from these arrhythmias — there was time to contemplate additional measures.
They had a history of non-ischemic cardiomyopathy (EF 30%), as well as PCI with one stent. In most middle-aged patients with a history of cardiomyopathy, a WCT will usually be VT. Fragmentation suggests scarring (ie, from prior MI and/or cardiomyopathy ). Furthermore, while specific criteria (e.g.
Instead — my thoughts were as follows: The rhythm is sinus , with marked bradycardia and a component of sinus arrhythmia. WPW Cardiac arrhythmias ( especially AFib ). Smith's — in that despite the alarming ST-T wave changes, I did not think ECG #1 was the result of an acute event. Abnormal ST-T wave abnormalities.
The chart revealed that the arrhythmia was not new. Atrial arrhythmias ( especially AFib or AFlutter ). It turned out the patient had cardiac amyloidosis. The presenting complaint was cough and fever from mild Covid pneumonia. There was no chest pain — and all troponins were negative. Prolonged QTc interval.
See this even more interesting and more dramatic and fascinating case: History of Hypertrophic Cardiomyopathy (HOCM), with Tachycardia and High Lactate = My Comment by K EN G RAUER, MD ( 10/28 /2023 ): = QUESTION: For clarity in Figure-1 — I've reproduced today's ECG without the long lead rhythm strip. Abnormal ST-T wave abnormalities.
By 1909 ECGs were being used to diagnose cases of arrhythmia; by 1910 to diagnose indicators of a heart attack. Another vendor to watch in this space is Israeli start-up CardiaCare, currently developing a ‘world-first’ closed-loop, neuromodulation wearable for the non-invasive treatment for AFib.
The absence of any wall motion abnormality makes ischemic cardiomyopathy very unlikely. The new onset cardiomyopathy was thought to be due to both drug/alcohol use and to Tachycardia-Induced Cardiomyopathy. Severe right ventricular enlargement and mildly decreased right ventricular systolic function. This is a “ generic ” term.
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