This site uses cookies to improve your experience. To help us insure we adhere to various privacy regulations, please select your country/region of residence. If you do not select a country, we will assume you are from the United States. Select your Cookie Settings or view our Privacy Policy and Terms of Use.
Cookie Settings
Cookies and similar technologies are used on this website for proper function of the website, for tracking performance analytics and for marketing purposes. We and some of our third-party providers may use cookie data for various purposes. Please review the cookie settings below and choose your preference.
Used for the proper function of the website
Used for monitoring website traffic and interactions
Cookie Settings
Cookies and similar technologies are used on this website for proper function of the website, for tracking performance analytics and for marketing purposes. We and some of our third-party providers may use cookie data for various purposes. Please review the cookie settings below and choose your preference.
Strictly Necessary: Used for the proper function of the website
Performance/Analytics: Used for monitoring website traffic and interactions
Given the irregular irregularity of beats #4-through 17 — Is this a run of AFib ( A trial Fib rillation ) with aberrant conduction? This raises the question if beats #4-thru-17 might represent a run of AFib with aberrant conduction? For more on fusion beats — See ECG Blog #128 and Blog #129 ). See ECG Blog #133 ).
MY Thoughts on the ECG in Figure-1: When faced with a challenging cardiac arrhythmia — It is a "luxury" to have access to a long lead rhythm strip containing 3 simultaneously -recorded leads. PEARL # 2: When the rate of AFib is rapid — this irregular tachycardia may look regular when it is not. QUESTIONS: Is the rhythm AVNRT or AVRT?
For more regarding ECG criteria for LVH — See the ADDENDUM below and/or ECG Blog #73 and ECG Blog #245. A bout H CM ( Different Forms of this Entity ): I've excerpted what appears below from My Comment in the December 26, 2023 post in Dr. Smith's ECG Blog. WPW Cardiac arrhythmias ( including AFib ).
NOTE: For more on ECG recognition of RVH and/or pulmonary hypertension ( re the qR pattern in lead V1 ) — See ECG Blog #234 and Blog #248. P utting I t A ll T ogether: At this point in my assessment of today's rhythm — I fully acknowledge that I did not know for certain the etiology of this arrhythmia.
I was sent the ECG shown in Figure-1 — being told only that providers on the case suspected AFib ( A trial Fib rillation ) with RBBB ( R ight B undle B ranch B lock ) aberrancy. As reviewed in ECG Blog #231 — QRS morphology in VT may manifest a number of different forms. How certain are YOU of your answer?
M Y T houghts on the ECG in Figure-1: I have presented similar ECGs to the one in today's tracing on several occasions ( most recently in ECG Blog #284 ). M y I MPRESSION : The rhythm in Figure -1 is almost certain to be very rapid AFib in a patient with WPW. The patient was hemodynamically stable in association with this rhythm. (
By the P s, Q s, 3 R Approach ( See ECG Blog #185 ): Regarding R egularity — the rhythm is irregularly irregular. As a result — IF the 1 lead you are monitoring happens to be one in which P waves are not well seen — then you might assume the irregular rhythm in front of you was AFib. ECG Blog #199 — for Review of M AT.
These findings suggest that instead of VT — the rhythm in Figure-1 is AFib with a fairly rapid ventricular response. Since the rhythm is supraventricular (ie, AFib ) — we can accurately assess QRS morphology. Given a lack of prior history — I don’t know if the AFib on ECG #1 is ( or is not ) a new finding.
What is unusual about this arrhythmia? NOTE: The ECG in Figure-1 has been recorded at the usual 25mm/second speed — but with the Cabrera format ( Please see my Editorial Note near the top of the page in ECG Blog #365 for review of the basics of this recording system ). ECG Blog #287 — More on AFlutter.
By the P s , Q s , 3 R Approach ( See ECG Blog #185 ): The rhythm in Figure-1 is clearly not R egular. PEARL #2: As cited in ECG Blog #252 — my favorite truism in arrhythmia interpretation is, "The commonest cause of a pause is a blocked PAC". ECG Blog #199 and ECG Blog #366 — for Review of M AT.
See our other blog posts of hypothermia and Osborn waves -- Massive Osborn Waves of Severe Hypothermia (23.6 Sci 5[4] 268-270, 2015 ) both highlight a likely association between acute development of ischemic J waves — and high risk of developing malignant ventricular arrhythmias ( My Comment in the September 23, 2020 post ).
NOTE: The ECGs in today's case are recorded in the Cabrera Format ( See Dr. Grauer Comment in the October 26, 2020 post of Dr. Smith's ECG Blog for review on the Cabrera Format ). Since the patient was stable and tolerating the arrhythmia it was decided to treat with IV Amiodarone for medical conversion. Smith : What do you think?
This blog explores the ways wearable technology can help track heart health, the advantages it offers, and how it contributes to better outcomes for those requiring surgical intervention. These early warnings are critical, as AFib increases the risk of stroke and other heart-related complications.
. = My Comment by K EN G RAUER, MD ( 3/15 /2023 ): = I found today’s case highly instructive in highlighting a number of important aspects regarding the presentation and initial treatment of a patient who presents to the ED with new AFib. I focus my comment on a few additional aspects regarding new AFib.
Is longterm endurance-training a risk factor for AFib and AFlutter? == Why is Today's Initial Rhythm AFlutter? The answer to this question is fascinating — albeit extending beyond the scope of this ECG Blog. Smith immediately say the rhythm was AFlutter with 1:1 AV conduction? Does Exercise Induce Non-Sinus Tachyarrhythmias?
My Comment , by K EN G RAUER, MD ( 7/5/2018 ): This blog post provides an excellent example of how a patient with SSS ( = S ick S inus S yndrome ) may present. during which sinus bradycardia and arrhythmia are seen but not to a degree that produces symptoms. New slow AFib reflects a combination of these rhythm problems.
This could be easily overlooked since there is complete heart block, but recognizing the atrial arrhythmia may mean prescribing anticoagulation to prevent stroke. As I discussed in detail in My Comment at the bottom of the page in the January 13, 2024 post in Dr. Smith's ECG Blog — pacemaker spikes tend to be a high frequency signal.
For more on Giant T waves — See My Comment at the bottom of the page in the June 22, 2020 and September 19, 2022 posts in Dr. Smith's ECG Blog ). WPW Cardiac arrhythmias ( including AFib ). Depth of the inverted T waves in leads V4 and V5 in Figure-1 attains this range. ( PEARL #1: Truly “giant” T waves are not overly common.
In some cases the ischemia can be seen "through" the flutter waves, whereas in other cases the arrhythmia must be terminated before the ischemia can be clearly distinguished. Learning Points: Acute arrhythmias such as SVT, rapid AF, and atrial flutter may coexist and/or be caused by ischemia, or vice versa.
Despite the irregularity of QRS complexes — this rhythm is not AFib — because at least some definite P waves are present ( RED arrows that I added at the bottom of ECG #1 ). The "usual rules" of cardiac arrhythmias are simply not always followed in critically ill patients.
I've copied KEY points from My Comment in the August 6, 2022 post in Dr. Smith's ECG Blog — regarding the answer to this question. Other Arrhythmias ( PACs, PVCs, AFib, Bradycardia and AV conduction disorders — potentially lethal VT/VFib ). Figure-1: The initial ECG in today's case. QTc prolongation.
As we often emphasize ( See My Comment in the May 5, 2020 post of Dr. Smith's ECG Blog ) — Statistically (ie, even before we look at the ECG itself ) — at least 80% of all regular WCT rhythms without clear sign of P waves will turn out to be VT ( V entricular T achycardia ). I'd add the following thoughts to Dr. Smith's discussion.
This was a very complex case and the details are too much for an ECG Blog, but suffice it to say that, s hortly thereafter, the patient had an asystolic arrest and was resuscitated. It will not always be possible to be 100% certain about the etiology of an arrhythmia from the single "snapshot" we get from a 10-second rhythm strip.
Therefore — the rhythm in ECG #1 is almost certain to be AFib ( A trial F ibrillation ) , seen here with a “rapid” ventricular response. Under normal conditions with AFib — the refractory period of the AV node does not allow more than 150-to-200 impulses/minute to be conducted to the ventricles.
The patient also has a history of AFib and HFmrEF ( = H eart F ailure with M inimally- R educed E jection F raction ). I have emphasized on many occasions in Dr. Smith's ECG Blog how AFlutter is by far (!) This patient presented to the ED “after a couple of days of chest discomfort”. Why was it Wrong to Think the Rhythm was AFlutter?
Patient was referred to electrophysiologic testing due to suspicion of afib and WPW. During electrophysiologic testing AVRT was induced, which degenerated to afib with ortho and antidromic conduction. Despite the near regularity in places — the reasons I immediately thought of WPW with very rapid AFib were i ) As per per Drs.
ECG 2 Especially in the context of the first ECG, readers of this blog will readily appreciate the ST elevations and hyperacute T waves in II, III, aVF, V6, and to a lesser extent V5. As discussed on this blog many times before, proportionality is key to the diagnosis of OMI by ECG. [link] I also texted the ECG to Dr. Smith.
Figure-2: A rapid method for estimating the QTc ( Figure from My Comment in the March 19, 2019 post in Dr. Smith's ECG Blog ). == Clinical Implications of a Short QTc: The differential diagnosis for today's tracing, with its short QTc ~360 msec. ) — but as can be seen, my estimate of ~360 msec. mg/dL — vs normal values = 4.4-5.2
C linical P oints R egarding E CG # 1 : We are told that the patient is a middle-aged woman and that she previously had been in AFib with LBBB. While I agree that AFib + complete AV block is the most likely rhythm diagnosis I'd like to see additional monitoring strips to be sure. Clinically What has happened in the interim?
The QRS is wide in B — but the rhythm is irregularly irregular with no sinus P waves — so this most probably represents rapid AFib with an atypical RBBB/LPHB morphology. We now see that QRS morphology in lead II during sinus rhythm is similar to the QRS morphology in lead II during rapid AFib (beats #1-5 in lead II in A).
Learning points : Takotsubo can lead to cardiac arrest from ventricular arrhythmia. I have periodically called attention to examples of the Ashman phenomenon as they occur in Dr. Smith's ECG Blog ( See My Comments in the January 5, 2020 post — the June 17, 2020 post — and the March 30, 2023 post , among others ).
MY THOUGHTS on ECG #1: My initial impression on looking at the ECG in Figure-1 — was that the rhythm was either rapid AFib in a patient with WPW — or — PMVT ( P oly M orphic VT ). The reason I initially thought the underlying rhythm was AFib — is that no atrial activity is seen in any lead and the rhythm “looks” irregular. See text ).
He has a family history concerning for arrhythmia. Given the circumstances of his car crash, we presume it was due to an underlying arrhythmia. He has a family history concerning for arrhythmia with his father requiring some sort of device (PPM, ICD, unclear) at a young age.
M Y A NSWER: The issue of whether C omputerized E CG I nterpretations are “at fault” for an inaccurate ECG diagnosis has been addressed numerous times on this blog. How can you avoid overlooking this arrhythmia? The reasons for overlooking this arrhythmia are simple: True MAT is not a common rhythm. GET a 12-lead!
(Time Window for Reperfusion; Acuteness on the ECG) == MY Comment by K EN G RAUER, MD ( 8/13/2020 ): == This is a challenging case — which is made all the more difficult by suboptimal quality of the initial 12-lead ECG — and lack of additional simultaneously-recorded leads for the arrhythmia tracings.
During hospital admission she had a variety of atrial arrhythmias, which eventually resolved, likely due to her decreasing flecainide level. For example: Statistical likelihood that the regular WCT in ECG #1 might be AFlutter ( instead of VT ) is greatly increased in a patient with AFib who is taking Flecainide.
Taking a Closer LOOK : There is a fairly marked sinus arrhythmia ( RED arrows in Figure-2 ). PEARL # 1: It is common with 2nd- and 3rd-degree AV block to see a "ventriculophasic" sinus arrhythmia. I've also carefully measured all P-P intervals in milliseconds — to illustrate the surprisingly marked sinus arrhythmia.
Instead — my thoughts were as follows: The rhythm is sinus , with marked bradycardia and a component of sinus arrhythmia. WPW Cardiac arrhythmias ( especially AFib ). Smith's — in that despite the alarming ST-T wave changes, I did not think ECG #1 was the result of an acute event. Abnormal ST-T wave abnormalities.
The chart revealed that the arrhythmia was not new. Extending beyond the scope of this emergency medicine ECG Blog — is that clinical and ECG manifestations of the various subtypes of cardiac amyloidosis may vary depending on the specific type of this deposited protein. Atrial arrhythmias ( especially AFib or AFlutter ).
With the history of Afib, CTA abdomen was ordered to r/o mesenteric ischemia vs ischemic colitis vs small bowel obstruction. We’ve presented many variations on this theme on Dr. Smith’s Blog — with today’s case being distinguished by its discovery on abdominal exam ! The patient had mild but diffuse abdominal tenderness.
Adenosine is safe in VT and may be useful in making the diagnosis. == MY Comment by K EN G RAUER, MD ( 12/23/2019 ): == It is ALWAYS great to welcome the contributions to Dr. Smith’s ECG Blog from Dr. Brooks Walsh — a highly skilled clinician + good friend and colleague who always stimulates conversation on important emergency medicine topics.
and if not — Is the rhythm “irregularly irregular”, as in AFib — or is there a pattern of “regular" irregularity in the form of group beating ? ). to diagnose almost any arrhythmia. What is the R ate? looking both at the atrial and ventricular rates IF these are different ). Is the rhythm R egular? (
WPW Cardiac arrhythmias ( especially AFib ). Tall R wave in lead V1 and/or early transition in the chest leads ( reflecting increased "septal" forces ). Abnormal ST-T wave abnormalities. Conduction defects (ie, LBBB, IVCD ). The Problem: None of the above ECG findings are specific for HCM.
Is This a Simple Right Bundle Branch Block? == MY Comment , by K EN G RAUER, MD ( 1/26/2020 ): == Dr. Smiths ECG Blog has presented too-numerous-to-count cases of hyperkalemia ( See My Comment in the 12/11/2018 post there are many others! ). Is this just right bundle branch block?
We organize all of the trending information in your field so you don't have to. Join thousands of users and stay up to date on the latest articles your peers are reading.
You know about us, now we want to get to know you!
Let's personalize your content
Let's get even more personalized
We recognize your account from another site in our network, please click 'Send Email' below to continue with verifying your account and setting a password.
Let's personalize your content