This site uses cookies to improve your experience. To help us insure we adhere to various privacy regulations, please select your country/region of residence. If you do not select a country, we will assume you are from the United States. Select your Cookie Settings or view our Privacy Policy and Terms of Use.
Cookie Settings
Cookies and similar technologies are used on this website for proper function of the website, for tracking performance analytics and for marketing purposes. We and some of our third-party providers may use cookie data for various purposes. Please review the cookie settings below and choose your preference.
Used for the proper function of the website
Used for monitoring website traffic and interactions
Cookie Settings
Cookies and similar technologies are used on this website for proper function of the website, for tracking performance analytics and for marketing purposes. We and some of our third-party providers may use cookie data for various purposes. Please review the cookie settings below and choose your preference.
Strictly Necessary: Used for the proper function of the website
Performance/Analytics: Used for monitoring website traffic and interactions
ECG#1 There is a regular tachycardia with a ventricular rate of about 180 bpm. Smith comment : When there is a regular wide complex tachycardia, first assess whether it is sinus or not. Put shortly is SVT with "Shark Fin STE" and not ventricular tachycardia. An ECG was recorded immediately and is shown below. Is there OMI?
Abstract Introduction Supraventricular tachycardia (SVT) is a common pediatric arrhythmia. Methods and Results This is a multicenter retrospective cohort study from three academic children's hospitals. Methods and Results This is a multicenter retrospective cohort study from three academic children's hospitals.
Wide-complex tachycardia: VT or aberrant, or "other?" A wide-complex tachycardia in an older patient must immediately suggest ventricular tachycardia. Sinus tachycardia with aberrancy was unlikely as the rate was consistently 150 bpm, without spontaneous variation. And indeed the QRS morphology strongly supported VT (e.g.
It's a very "fun" ECG, with initial ectopic atrial tachycardia (negative P waves in inferior leads conducting 1:1 with the QRSs), followed by spontaneous resolution to sinus rhythm. What About the Tachycardia? Today's regular SVT rhythm could be an ectopic atrial tachycardia ( ATach ) — or an automatic junctional tachycardia.
The above ECGs show the initiation and continuation of a polymorphic ventricular tachycardia. Polymorphic ventricular tachycardia can be ischemic, catecholaminergic or related to QT prolongation. This distinction is more than academic — because both treatment and the response to therapy tend to be different with these 2 entities.
Multifocal Atrial Tachycardia 2. M Y A NSWER: In my opinion — it is both academic and clinically unimportant ( as well as often impossible ) to attempt to distinguish between sinus rhythm with multiple different-looking PACs vs MAT. The rhythm is indeed irregularly irregular, so atrial fibrillation must be considered.
A 50-something male with unspecified history of cardiomyopathy presented in diabetic ketoacidosis (without significant hyperkalemia) with a wide complex tachycardia and hypotension. Analysis: there is a wide complex tachycardia. This was the interpretation I put into the system: WIDE COMPLEX TACHYCARDIA. It is regular.
In all probability, this dilation is a form of atrial tachycardia and atrial cardiomyopathy. Spatial relationship of sites for atrial fibrillation drivers and atrial tachycardia in patients with both arrhythmias July 2017 International Journal of Cardiology 248(3) AF begets AF. Is there proof for this, or just an academic gossip?
This usually represents posterior OMI, but in tachycardia and especially after cardiac arrest, this could simply be demand ischemia, residual subendocardial ischemia due to the low flow state of the cardiac arrest. This prompted cath lab activation. On arrival to the ED, this ECG was recorded: What do you think?
The shortened PR-interval, specifically, proved to be quite beguiling as it swept crews down a differential diagnosis of intermittent accessory pathway syndrome – insomuch as a “syndrome” of recurrent tachycardia to account for the patient’s symptoms. Thus, a high index of suspicion is paramount.
That said — rhythm interpretation here is more of an academic interest — as the most striking findings are the widespread T-wave inversions and QT prolongation. But there are 3 other wide beats in the tachycardia that begins with beat #6 ( = beats #7; 13,14 ). The patient was diagnosed with stress cardiomyopathy.
A useful classification of WCT ( W ide- C omplex T achycardia ) rhythms — separates them into those that are mono morphic ( with similar QRS morphology during the tachycardia ) vs those that are poly morphic ( in which QRS morphology varies ). PEARL #2 — Distinction between PMVT vs Torsades is more than academic.
I find AV dissociation in VT to be very difficult to differentiate from artifact, as there are always random blips on tachycardia tracings. Read this post: Idiopathic Ventricular Tachycardias for the EM Physician 2. Definitive diagnosis that ECG #1 is in fact VT is more than academic. Ken notes AV dissociation.
2) Tachycardia to this degree can cause ST segment changes in several ways. First , there can simply be diffuse ST depressions (which obligates reciprocal STE in aVR) associated with tachycardia which are not indicative of ischemia. Sometimes you must correct the rhythm to see what lies underneath. Is this inferor STEMI?
There is sinus tachycardia (do not be fooled into thinking this is VT or another wide complex tachycardia!) From an academic standpoint — I found several subtle ECG findings from the 2 tracings in this case to be especially interesting. She was alert and oriented and hypotensive with initial BP 70/50. Eur Heart J.
This progressed to electrical storm , with incessant PolyMorphic Ventricular Tachycardia ( PMVT ) and recurrent episodes of Ventricular Fibrillation ( VFib ). Distinction of PMVT vs VFib is an academic one in this case ). The patient improved, and on Day-11 of the hospital stay — he was off inotropes and on a small dose of a ß-blocker.
If the patient has Abnormal Vital Signs (fever, hypotension, tachycardia, or tachypnea, or hypoxemia), then these are the primary issue to address, as there is ongoing pathology which must be identified. Academic Emergency Medicine., Academic Emergency Medicine, 2003 Volume 10, Number 5 539-540. Thiruganasambandamoorthy, V.,
2025.110515 [link] Abstract Objective: The administration of amiodarone or lidocaine is recommended during the resuscitation of out-of-hospital cardiac arrest (OHCA) patients presenting with defibrillation-refractory or recurrent ventricular fibrillation or ventricular tachycardia. DOI: 10.1016/j.resuscitation.2025.110515
We organize all of the trending information in your field so you don't have to. Join thousands of users and stay up to date on the latest articles your peers are reading.
You know about us, now we want to get to know you!
Let's personalize your content
Let's get even more personalized
We recognize your account from another site in our network, please click 'Send Email' below to continue with verifying your account and setting a password.
Let's personalize your content