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Sent by anonymous, written by Pendell Meyers, reviewed by Smith and Grauer A man in his 40s presented to the ED with HTN, DM, and smoking history for evaluation of acute chestpain. He was eating lunch when he had sudden onset chest pressure, 9/10, radiating to his back, with sweating and numbness in both hands.
Click here to sign up for Queen of Hearts Access Case A 58-year-old woman presented to the ED with burning chestpain that started 2-3 hours earlier while sitting on a porch swing. In any case, it is diagnostic of OMI in a chestpain patient. But there is also perhaps some STD in inferior leads -- this would support LAD.
Written by Jesse McLaren Three patients presented with acute chestpain and ECGs that were labeled by the computer as completely normal, and which was confirmed by the final cardiology interpretation (which is blinded to patient outcome) also as completely normal. What do you think?
A 50-something man presented in shock with severe chestpain. We recorded an ECG in which V1-V3 were put in the position of V4R-V6R, and V4-6 were placed in V7-9 to (academically) confirm posterior OMI. I say academically because the STD in V2 is diagnostic -- posterior leads are NOT necessary. What to do?
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Submitted and written by Anonymous, edits by Meyers and Smith A 50s-year-old patient with no known cardiac history presented at 0045 with three hours of unrelenting central chestpain. The pain was heavy, radiated to her jaw with an associated headache. Academic Emergency Medicine 27(S1): S220. Abstract 556.
He then went on to say: "40-something with chestpain for one hour. Burning pain subxiphoid and into throat." I’ll emphasize that the generalities stated here may not pertain early on in the process in a patient with a worrisome history of new-onset chestpain. Had episode of nausea and dizziness when it started.
It is from a 50-something with chestpain: What do you think? I first became interested in computerized ECG interpretation in the beginning of my academic days in the early 1980s ( References to some of my work appear below — as I believe I may have been the first family physician to publish in this area ).
A 67 yo f developed chestpain this morning." See this case: A man his 50s with chestpain. Academic Emergency Medicine 27(S1): S220; May 2020. The Need for Prompt Cath: — Way # 1 — In a 67-year old patient with several hours of new chestpain — ECG #1 clearly fits the description of deWinter T waves.
Dr. Stone is Director of Academic Affairs for the Mount Sinai Health System and Professor of Medicine (Cardiology) and Professor of Population Health Science and Policy at the Icahn School of Medicine at Mount Sinai, and the study chair of the ECLIPSE trial. These results extend the strong recommendations from recent U.S.
This was written by Magnus Nossen, from Norway, with comments and additions by Smith A 50 something smoker with no previous medical hx contacted EMS due to acute onset chestpain. Upon EMS arrival the patient appeared acutely ill and complained of chestpain. Of academic interest — are the arrhythmias that developed.
See this case: Persistent ChestPain, an Elevated Troponin, and a Normal ECG. This is different from nitroglycerin which produces vasodilation and can improve by pain improving myocardial perfusion. You do not need to be better than the Queen of Hearts at EKG to understand that refractory chestpain NEEDS CATH NOW.
This means that at every age, the probability a man complaining of chestpain has significant underlying coronary disease as a cause of this chestpain is much higher than a woman complaining of chestpain. Thanks for reading Dr. The data is overwhelming every way you can possibly look at it.
A man in his 70s with past medical history of hypertension, dyslipidemia, CAD s/p left circumflex stent 2 years prior presented to the ED with worsening intermittent exertional chestpain relieved by rest. This episode of chestpain began 3 hours ago and was persistent even at rest. Troponin was ordered.
A middle-aged woman had an acute onset of chestpain and dyspnea. The pain had almost resolved by the time an ECG was obtained in the ED: Here is the computer diagnosis What do you think? In the interest of academic discussion — I’ll add the following thoughts: The computer interpretation was obviously wrong.
Cardiac Cath labs waiting for some major influx of COVID heart damage not only didn’t see patients presenting with COVID heart attacks, but they idled as patients terrified of coming to the hospital stayed home rather than come to the hospital with chestpain.
With more than 500 peer-reviewed publications, the HeartFlow FFR CT Analysis remains unparalleled in precision coronary care, as supported by the ACC/AHA ChestPain Guidelines, to improve treatment plans and outcomes. Simon, MD , President, Academic & External Affairs and Chief Scientific Officer, University Hospitals Health System.
Written by Willy Frick A 50 year old man with no medical history presented with acute onset substernal chestpain. His ECG is shown below. Pretty obvious anterior current of injury. This was a machine read STEMI positive OMI. Readers of this blog can easily appreciate the hyperacute T waves in the precordium, clearest in V1-V4.
He had concurrent sharp substernal chestpain that resolved, but palpitations continued. Over past 3 months, he has had similar intermittent episodes of sharp chestpain while running, but none at rest. Definitive diagnosis that ECG #1 is in fact VT is more than academic.
Here are some cases of RBBB with LAFB: What is the Diagnosis in this 70-something with ChestPain? Some patients have baseline RBBB with LAFB, but in patients with likely ACS, these are associated with severe infarction with cardiac arrest, cardiogenic shock or impending shock.
Data were derived from the American College of Cardiology ChestPain – MI Registry data collected by trained abstractors at the facility and were verifed by the study team.
This was sent by : Jacob Smith, DO Emergency Medicine Resident Ohio Health Doctors Hospital Emergency Residency Christopher Lloyd, DO, FACEP Director of Clinical Education, USACS Midwest Case A 30 year old patient presents to triage with chestpain. link] Here is the history: A 30 yo man presented complaining of severe chestpain.
This is one case where it made a difference: Right Ventricular MI seen on ECG helps Angiographer to find Culprit Lesion Nevertheless, it is sometimes a fun academic exercise to try to predict the infarct artery: An elderly patient had onset of chestpain one hour prior. He called 911. Here is the prehospital ECG.
Other cases of LAD OMI with RBBB/LAFB: A man in his 40s who really needs you to understand his ECG Cardiac Arrest at the airport, with an easy but important ECG for everyone to recognize A woman in her 60s with 6 hours of chestpain, dyspnea, tachycardia, and hypoxemia Ventricular Fibrillation, ROSC after perfusion restored by ECMO, then ECG.
The chestpain quickly subsided. This distinction is more than academic — because both treatment and the response to therapy tend to be different with these 2 entities. The T-waves have more area under the curve than baseline, but don't appear like typical hyperacute T waves. Are you worried about OMI in this case?
This is a 58 year old male with 40 minutes of chestpain of acute onset. He was given aspirin and sublingual nitroglycerine, which improved his pain. Published in Academic Emergency Medicine, vol. The cath lab was activated by the paramedics. On arrival, the following ECG was recorded. 18 (5 Suppl 1):Abstract 425, p.
The patient in today’s case is a previously healthy 40-something male who contacted EMS due to acute onset crushing chestpain. The pain was 10/10 in intensity radiating bilaterally to the shoulders and also to the left arm and neck. Distinction of PMVT vs VFib is an academic one in this case ).
Sent by anonymous, edited by Pendell Meyers A man in his 50s with history only of hypertension presented with acute chestpain that started 45 minutes prior to presentation while doing yard work. Academic Emergency Medicine 27(S1): S220; May 2020. Setting – large, academic, suburban ED. Abstract 556.
Factors consistently manifesting as such, in addition to chestpain, include, diaphoresis, vomiting, radiation of pain (most alarming when inclusive of both arms), and pain aggravated by exertion. [1] It should be emphasized here that this is a presentation of high-pretest probability for Acute Coronary Syndrome (ACS).
This 60-something with h/o COPD and HFrEF (EF 25%) presented with SOB and chestpain. M Y A NSWER: In my opinion — it is both academic and clinically unimportant ( as well as often impossible ) to attempt to distinguish between sinus rhythm with multiple different-looking PACs vs MAT. Here is the ECG: What do you think?
More evidence this past week that vaccine myocarditis is very much a real entity while COVID19 myocarditis is mostly a fabrication of academic researchers comes from Scandinavian countries. Subscribe for free to receive new posts and support my work. Notice that there is no spike in myocarditis diagnoses until the second half of 2021.
A recent similar case: A 40-something with chestpain. PEARL #2 — Distinction between PMVT vs Torsades is more than academic. This is commonly found after epinephrine for cardiac arrest, but could have been pre-existing and a possible contributing factor to cardiac arrest. Is this inferior MI?
It is interesting particularly because the ensconced narrative in academic circles and the medically related twitterati that seem to have an outsized influence on COVID related policy has been that vaccine myocarditis is “mild” Leo Lam, Ph.D.💉⚕️🌎🇺🇸🇬🇧🏴🇭🇰
Check : [vitals, SOB, ChestPain, Ultrasound] If the patient has Abdominal Pain, ChestPain, Dyspnea or Hypoxemia, Headache, Hypotension , then these should be considered the primary chief complaint (not syncope). Academic Emergency Medicine., Academic Emergency Medicine, 2003 Volume 10, Number 5 539-540.
That's the only way we can give women a definitive diagnosis for what's causing their chestpain." The paper specifically notes that it operates "differently than some traditional prevention centers because it assesses for obstructive as well as nonobstructive causes for IHD."14
He contacted EMS due to acute onset chestpain and feeling unwell and fatigued. He subsequently developed worsening chestpain. This, in the context of worsening chestpain , is evidence of reocclusion of the infarct-related artery and active OMI in development. See this case: A man his 50s with chestpain.
This was texted to me by a paramedic while I was out running one day: "54 yo male chestpain started at 1pm. History of diabetes type II and stent placement in 2018. I’m seeing hyperacute T waves III, aVF, down sloping depression I and aVL. What do you think? I responded: "Definite inferior OMI. And Right Ventricular.
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