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Wide Complex Tachycardia converted, subsequent 12-lead with ST elevation due to WPW

Dr. Smith's ECG Blog

One might think this represents acute STEMI, or Bundle branch block with discordant ST segments and suspicously concordant T-waves. Ablation was planned for a later date. Upon arrival to the ED, he had the following 12-lead ECG: There is striking ST segment elevation in V1 and V2, with ST depression in V3-V6 as well as I, II, and aVF.

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What are treatment options for this rhythm, when all else fails?

Dr. Smith's ECG Blog

The ECG shows obvious STEMI(+) OMI due to probable proximal LAD occlusion. In such cases — radiofrequency ablation of ectopic beats triggering malignant ventricular arrhythmias was needed for control of arrhythmic storm because the antiarrhythmic medications tried were ineffective ( Marrouche et al — JACC 5;43(9): 1715-20, 2004 ).

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What is the rhythm? And is there new left bundle branch block (LBBB)?

Dr. Smith's ECG Blog

In other words, after reperfusion therapy for STEMI, the appearance of AIVR is usually a good sign, meaning that the artery is reperfused. Our electrophysiologist, Rehan Karim, states he has ablated AVNR"T" ("T" because it is not tachycardia) in a 90 year old, and that he has seen rate-related BBB at very slow rates.

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A 30-something woman with intermittent CP, a HEART score of 2 and a Negative CT Coronary Angiogram on the same day

Dr. Smith's ECG Blog

A 34 yo woman with a history of HTN, h/o SVT s/p ablation 2006, and 5 months post-partum presented with intermittent central chest pain and SOB. Thus, Wellens' syndrome should be thought of as a transient OMI or transient STEMI. Transient STEMI is at high risk of re-occlusion. This was sent by a colleague.

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Chest pain and anterior ST depression. What’s the cause(s)?

Dr. Smith's ECG Blog

But it doesn’t meet STEMI criteria, and was not identified by the computer or the over-reading cardiologist. Still no WPW pattern, and more obvious inferoposterior OMI, but still STEMI negative. The emergency physician wasn’t sure what to make of the changes from one ECG to the next but was concerned about ACS. What do you think?

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Why many Ischemic VTs are not abolished by revascularization ?

Dr. S. Venkatesan MD

There is a problem here too ,even critical Ischemia with high grade unstable angina rarely trigger a VT but STEMI seems to have the exclusive rights to trigger it , by its ability to produce acute transmural ischemia. best , it is just a back up device to tackle the escaped VTs in spite of RF ablation and drugs. Reference 1.