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First runner-up for the YIA is Seokhun Yang, MD , of Seoul National University Hospital, for his abstract, “Prognostic Implications Of Coronary CT Angiography-derived Plaque And Hemodynamic Features On Acute Coronary Syndrome Across Varying Time Intervals: Emerald-ii Study.” and the Ma Family, who provided a $5,000 case prize for the winner.
Stroke, Volume 56, Issue Suppl_1 , Page AWP193-AWP193, February 1, 2025. Plaques were segmented on PD images from each time point. Plaques were compared between consecutive time points and classified as progressing, stable, or regressing, based on volume change (those <5mm3were excluded).
Stone, MD Mount Sinai Health System tim.hodson Wed, 04/02/2025 - 15:26 March 31, 2025 Using intravascular imaging (IVI) to guide stent implantation during complex stenting procedures is safer and more effective for patients with severely calcified coronary artery disease than conventional angiography, the more commonly used technique.
Stroke, Volume 56, Issue Suppl_1 , Page ATP358-ATP358, February 1, 2025. Most dementia patients exhibit mixed brain pathologies, with histological evidence of ischemia and A plaque accumulation, observed at autopsy. Introduction:Stroke is a well-established independent risk factor for the development of dementia.
There is low voltage in the precordium which always makes reading ischemia harder. In ACS, chest pain is the warning sign of ongoing ischemia. Smith : As Willy says, and as we've said many times before, morphine will resolve pain without resolving ischemia. ECG 1 What do you think? To me, this ECG is not diagnostic.
Influenza-like illness can also trigger plaque rupture. McLaren : The inferior T wave inversion suggests either reperfusion (if resolved symptoms) or subacute refractory ischemia and from the previous description of pain refractory to nitro it is likely the latter.
Time 7 hours lead reversal There is limb lead reversal (QRS in I and aVL are now inverted), but nevertheless one can see that the ischemia appears to have resolved. This was attributed to a "Type 2 MI", which is acute MI that is not due to ruptured plaque, but rather due to "supply demand oxygen mismatch". Next day, with K = 4.6
It is possible there is microvascular dysfunction producing residual transmural ischemia. But this is most common when there is prolonged ischemia, and this patient had the fastest reperfusion imaginable! Mechanisms of plaque formation and rupture. Coronary plaque disruption. Journal of Geriatric Cardiology , 19 (6).
That said there were no clinical symptoms or ECG findings suggestive of ongoing ischemia. There were no plaques or stenoses. You have given IV MgSO4 a fast acting -blocker and IV amiodarone bolus and infusion. The possibility of an ischemic cause of the ventricular arrhythmia has to be considered! 3] Lavalle, C.
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