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An echocardiogram at 13:40 showed: Severely reduced global systolic function with an estimated EF of 10-20% Mildly increased LV size Akinesis of the entire septum and apex Hypokinesis of the anterior, anterolateral, and mid posterior segments A final troponin T was drawn at 17:23- 3,475 ng/L.
Long term outcome is not available. == MY Comment, by K EN G RAUER, MD ( 2/1/2025 ): == We need to learn from cases like today's. This distinction is further complicated because marked LVH may at times mask the ST-T wave changes of acute ischemia. Here are some images: Next morning ECG: Reperfusion findings are clear.
The patient was started on heparin for possible NSTEMI vs demand ischemia. increasing stenosis, ischemia, volume changes, increased blood pressure, atrial fibrillation, etc.) The EKGs from the ED presentation were felt by cardiology to represent "subendocardial ischemia." Smith : these ECGs do NOT show subendocardial ischemia.
In this study of consecutive patients with LBBB who were hospitalized and had an echocardiogram, a QRS duration less than 170 ms (n = 262), vs. greater than 170 ms (n = 38), was associated with a significantly better ejection fraction (36% vs. 24%). Negative trops and negative angiogram does not rule out coronary ischemia or ACS.
That said there were no clinical symptoms or ECG findings suggestive of ongoing ischemia. It is reasonable to perform an echocardiogram to evaluate LV function. You have given IV MgSO4 a fast acting -blocker and IV amiodarone bolus and infusion. The possibility of an ischemic cause of the ventricular arrhythmia has to be considered!
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