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Coronary artery spasm (CAS), or Prinzmetal angina, is a recognised cause of myocardial ischaemia in non-obstructed coronary arteries which typically presents with anginal chestpain. The patient presented with recurrent palpitations and pre-syncope, with no chestpain.
A 54-year-old male athlete was evaluated at this hospital because of exertional dyspnea and chestpain. Physical examination revealed jugular venous distention and Kussmauls sign. A diagnosis was made.
Written by Jesse McLaren A 50 year old presented to triage with one hour of chestpain, and the following ECG labeled normal by the computer (GE Marquette SL) algorithm. CJEM 2025 March 10 3. Please see My Comment at the bottom of the page in the January 15, 2025 post for "My Take" on what constitutes a "normal" ECG ).
tim.hodson Tue, 03/04/2025 - 14:11 Feb. each year, according to the American Heart Association 2025 Heart Disease and Stroke Statistical Update. 22, 2025More than 60,000 people die from heart valve disease (HVD) in the U.S. AS occurs when the aortic valve narrows, restricting blood flow from the heart to the body.
tim.hodson Fri, 03/28/2025 - 15:23 Mar. TheNational Institute for Health and Care Excellence(NICE) recommends CCTA as the first-line investigation for patients with chestpain due to suspected CAD, highlighting its importance in improving diagnostic certainty.
Second runner-up is Marcel Langenbach, MD for his abstract, "Pericoronary Adipose Tissue Density Relates To Increased Cardiovascular Adverse Events In Patients With Stable ChestPain: Insights From The PROMISE Trial." The SCCT Best Abstract Award is supported by the Cardiovascular Research Foundation of Southern California (CVRF of So.
Stone, MD Mount Sinai Health System tim.hodson Wed, 04/02/2025 - 15:26 March 31, 2025 Using intravascular imaging (IVI) to guide stent implantation during complex stenting procedures is safer and more effective for patients with severely calcified coronary artery disease than conventional angiography, the more commonly used technique.
A 50-year-old man presented to the emergency department with symptoms of acute chestpain, dizziness, and headache. If the dissection extends into the aortic arch branches, ensuring adequate cerebral perfusion during surgery is crucial to preventing stroke. His blood pressure was 180/110 mmHg and heart rate was 100 bpm.
Stroke, Volume 56, Issue Suppl_1 , Page AWP141-AWP141, February 1, 2025. In unadjusted analysis, factors significantly associated with troponin testing were a triage complaint of chestpain, older age, higher mean systolic BP, hypertension, diabetes, obesity, stroke or TIA, congestive heart failure, or coronary disease.
Noel Bairey Merz, MD , director of the Barbra Streisand Women's Heart Center , will participate in a cardiovascular health for women session, “Where Are We Now: From WISE to CHESTPAIN Guidelines,” Sunday, April 7, 12:45-12:52 p.m. 24 that will bring together participants in the 2025 Clinical Trials Research (CTR) program.
He contacted EMS due to acute onset chestpain and feeling unwell and fatigued. He subsequently developed worsening chestpain. This, in the context of worsening chestpain , is evidence of reocclusion of the infarct-related artery and active OMI in development. See this case: A man his 50s with chestpain.
He was given two separate sprays of nitroglycerin sublingually, neither of which improved his pain but did cause him to become briefly hypotensive ( 600 ng/L. The patient has acute persistent refrectory chestpain and elevated troponin. There is no need for another ECG. Just go to the cath lab!
Written by Hans Helseth A 34 year old man with no known medical history presented to the ED after an hour of chestpain. He described the pain as a mid sternal "burning sensation" and rated it 8.5 out of 10 at onset, but on presentation to the ED, reported that the pain had improved to 4.5. 10 chestpain.
Case A 76 year old man with chronic hypertension but no history of coronary disease or myocardial infarction presented to the ED with chestpain at 2343. It is awaiting FDA approval (but approved for 1.5 There is ST elevation in the inferior leads. The Queen of Hearts recognizes this as OMI ("STEMI/STEMI Equivalent").
Scenario 1 : The patient presents with 24 hours of substernal chestpain. Ninety percent of patients with reperfusion attained a maximum T wave negativity of 3 mm or more within 48 hours after the onset of chestpain in the lead that initially displayed the greatest ST segment elevation. Below is his presentation ECG.
A 50-something male presented to triage with chestpain for one day. A Chest X-ray showed infiltrates. Thus, another etiology of chestpain is found, and the fever suggests "fever-induced Brugada." The presenting complaint noted at Triage was, "a 50yo man with chestpain!" The temperature was 39.5
She did not even need to ask in this case, because even if the patient presented with chestpain, she would call it NEGATIVE. Fortunately, the physicians seeing the patient were using the Queen of Hearts PM Cardio AI ECG OMI Model, and she had absolutely no concerns.
The patient received three nitroglycerin tablets with significant "improvement" in his chestpain. Improved chestpain is unresolved chestpain. I am commonly told, and I commonly read in the chart that chestpain is resolved. It is not enough for the chestpain to be "much better."
Written by Pendell Meyers An adult man presented with acute chestpain. Acute chestpain, right bundle branch block, no STEMI criteria, and negative initial troponin. What is the Diagnosis in this 70-something with ChestPain? 68 minutes with chest compressions, full recovery.
A 50 something male presented in the evening to ED for evaluation of chestpain that started at 1600. Note: the 2022 ACC Expert consensus Chestpain guidelines state that "posterior STEMI-Equivalent" is a sign of acute coronary occlusion. The chestpain continued for hours. hours, another ECG was recorded.
He stated later that he gave her 1 sublingual NTG and her pain went down to 1/10. This 60-year old woman clearly presented to the ED as a higher -risk patient given her history of known coronary disease, now with new chestpain. He did not repeat an ECG. His response: "Yeah, thats what I was afraid of.
A 30 year old African American Male presented to the ED with chestpain that occurred the day before. There was no pain on the day of presentation. 2019.06.007) (Full text here: [link] ) The ECG above is diagnostic of Wellens' syndrome (full reference below): 1) Episode of anginal chestpain that is resolved (GONE!)
No chestpain. Im changed. == MY Comment , by K EN G RAUER, MD ( 1/16 /2025 ): == It's not often that we see a clinical entity for which it seems that the patient "read the textbook" before the ECG was recorded. hs Trop I is 15,000. I think it is OMI. Taking her to the cath lab. He wrote: OMI it was. Large diagonal. Now opened.
Written by Jesse McLaren An 80 year old with a history of CHF, ESRD on dialysis, and multiple prior cardiac stents presented to the emergency department with 3 days of intermittent chestpain and shortness of breath that resolved after nitro, which felt like prior episodes of angina. The patient had no further episodes of chestpain.
Written by Pendell Meyers, sent by anonymous, with additions by Smith A man in his 40s had acute chestpain and called EMS. 3) [link] Artificial intellingence based detection of acute coronary occlusion compared to STEMI criteria - External validation study in a consecutive all-comer German chestpain unit cohort.
A 69 year old woman with a history of hypertension presented to the emergency department by EMS for evaluation of chestpain and shortness of breath. She awoke in the morning with sharp chestpain which worsened throughout the morning. As her pain worsened, so did her dyspnea. This was written by Hans Helseth.
The patient contacted the ambulance service after he experienced sudden onset chestpain and diaphoresis that had started 20 minutes prior. The above ECG is from man in his 80s with crushing chestpain. New England Journal of Medicine. == MY Comment, by K EN G RAUER, MD ( 1/26/2025 ): == "Some patients read the textbook.
Written by Willy Frick A man in his 60s with a history of hypertension and 40 pack-year history presented to the ER with 1 day of intermittent, burning substernal chestpain radiating into both arms as well as his back and jaw. It has been stuttering, lasting 10 minutes at a time with associated diaphoresis.
Written by Willy Frick A woman in her 60s with very severe hyperlipidemia (LDL >200 mg/dL) presented with acute onset chestpain. She described the pain as moderate in severity, and said it had come and gone several times over the next few hours before ultimately resolving. Her symptoms began while getting off the bus.
The ECG in Figure-1 was obtained from a middle-aged man who presents to the ED ( E mergency D epartment ) with 6 hours of chestpain. Figure-1: The initial ECG in today's case obtained from a middle-aged man with 6 hours of chestpain. ( He is hemodynamically stable. They lead you to numerous posts with more on OMIs.
He denied chestpain. It is correct that he did not have chestpain, but we must remember that fully 1/3 of full blown STEMI do not present with chestpain. I read them without clinical context and looked at the clinical context later. Bedside POCUS showed very poor LV function and a few pulmonary B lines.
PEARL # 2: Did you notice in the post-conversion tracing that there is ST segment coving with fairly deep T wave inversion in the chest leads? Clinical correlation, and possibly Troponin levels are needed to determine whether this deep T wave inversion needs additional evaluation ).
A 62 year old man with hyperlipidemia presented to a rural emergency department with 7 hours of 3/10 chestpain. 5 years later ( now in 2025 ) the problem remains. In fact, much of what passes for EKG education can actually harm one's interpretation skills.
This is a very bold statement in a type 1 diabetic with very concerning sounding chestpain. The patient was treated with aspirin and a GI cocktail, which did not help the pain. The emergency medicine physician documented, "His initial EKG is riddled with artifact and difficult to interpret but does not look like a STEMI."
Written by Pendell Meyers A man in his 60s presented with acute chestpain. Long term outcome is not available. == MY Comment, by K EN G RAUER, MD ( 2/1/2025 ): == We need to learn from cases like today's. Here is his triage ECG: What do you think? There is sinus rhythm with clear LVH.
With OMI, all you know is that your patient has some nonspecific chestpain, SOB, shoulder pain etc. First, When you have a rhythm problem, you know you have a problem because the rate is either fast, slow, or irregular. which is probably NOT due to acute MI.
He had no chestpain, dyspnea, or any other anginal equivalent, and his vital signs were normal. The cardiologist thought she had stent thrombosis which is possible, but I do not necessarily think is sufficient to explain her complete hemodynamic collapse. & Dawson, D. Circulation , 145 (13), 10021019.
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