Dr. Smith's ECG Blog

APRIL 13, 2025

There is also STE in V1 which is diagnostic of right ventricular OMI in this situation , and partly explains the syncope and hypotension (along with the bradycardia). I suspect this statement is still true 2 years later, in 2025. These plaques will often not be recognized as "culprits", because no fissuring or ulcertaion is seen.

Chest Pain 84

Chest Pain Embolism STEMI Plaque 84

Dr. Smith's ECG Blog

MARCH 19, 2025

Influenza-like illness can also trigger plaque rupture. Despite the baseline artifact theres sinus bradycardia, convex ST elevation in III, reciprocal ST depression in aVL and possible anterior ST depression indicating inferoposterior OMI. Beware confusing the diagnosis of posterior STEMI by using posterior leads.

Chest Pain 116

Chest Pain STEMI Stents Stent 116

Dr. Smith's ECG Blog

FEBRUARY 6, 2025

There are 2 main options: Overdrive pacing could be considered and in the right clinical situation, this is often effective for reducing ventricular arrhythmias ( especially in the case of preventing pause induced or bradycardia-induced arrhythmias in association with QTc prolongation ). There were no plaques or stenoses.

Tachycardia 72

Tachycardia Arrhythmia Ablation Atrial Flutter 72

Dr. Smith's ECG Blog

MARCH 8, 2025

Within ten minutes, she developed bradycardia, hypotension, and ST changes on monitor. Bradycardia and heart block are very common in RCA OMI. Mechanisms of plaque formation and rupture. Coronary plaque disruption. He told the patient this horrible news. The other point in favor of RCA is junctional rhythm. Virmani, R.,

Cardiomyopathy 75

Cardiomyopathy Myocardial Infarction Defibrillator Bradycardia 75