Remove 2025 Remove Arterioles Remove Strokes
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Abstract TMP5: The Impact of Enteral Nimodipine on Endothelial Cell Apoptosis in an Animal Subarachnoid Hemorrhage Model

Stroke Journal

Stroke, Volume 56, Issue Suppl_1 , Page ATMP5-ATMP5, February 1, 2025. In penetrated arterioles, the expression of Bax in the SAH-nimodipine group was significantly lower than that of the SAH-saline group (p < 0.001).

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Abstract WP60: Orosomucoid Expression in Cerebral Amyloid Angiopathy

Stroke Journal

Stroke, Volume 56, Issue Suppl_1 , Page AWP60-AWP60, February 1, 2025. Background:Cerebral amyloid angiopathy (CAA), in which amyloid- (A) is deposited in cerebral and meningeal blood vessels, is associated with not only stroke but also cognitive dysfunction.

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Abstract TMP63: Genetic investigation for cerebral small vessel disease genes in patients with spontaneous intracerebral hemorrhage

Stroke Journal

Stroke, Volume 56, Issue Suppl_1 , Page ATMP63-ATMP63, February 1, 2025. Objectives:Spontaneous intracerebral hemorrhage (ICH) is caused by the rupture of small arterioles due to cerebral small vessel disease (SVD), commonly with hypertensive arteriolosclerosis (HTA) or cerebral amyloid angiopathy (CAA).

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Abstract TMP111: Perivascular von Willebrand factor (VWF) promotes irregular remodeling of leptomeningeal collaterals in response to sustained increased luminal flow.

Stroke Journal

Stroke, Volume 56, Issue Suppl_1 , Page ATMP111-ATMP111, February 1, 2025. We previously demonstrated abnormal deposition of VWF in themurallayer of leptomeningeal collateral arterioles (LMCs) during flow-induced outward remodeling. Introduction:VWF is an endothelial protein with known roles in hemostasis and thrombosis.