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A CT Coronaryangiogram was ordered. Here are the results: --Minimally obstructive coronary artery disease. --LAD Although a lesion is not visible anatomically on this CT scan, coronary catheter angiography could be considered based on Cardiology evaluation." A repeat troponin returned at 0.45 CAD-RADS category 1. --No
From afar, there is gross tachycardia, cadence irregularities, and narrow QRS complexes that may, or may not, be Sinus in origin; and finally – a cacophony of wide complexes that might very well be ventricular in origin. McLaren : We’ve answered the first question – Sinus Tachycardia with episodic runs of wide QRS (RBBB morphology) and PVC’s.
Ct coronaryangiogram showed normal coronary arteries. Smith note: I think CT coronaryangiogram is reasonable with the elevated troponins and symptoms. T-wave inversions and dynamic ST elevation Tachycardia, hyperthyroid, and ST elevation. He was diagnosed with mild AKI which resolved. What is it?
We investigated the incidence of an acutely occluded coronary in patients presenting with STE-aVR with multi-lead ST depression. All electrocardiograms (ECGs) and coronaryangiograms were blindly analyzed by experienced cardiologists. A slightly prolonged QTc ( although this is difficult to assess given the tachycardia ).
I find AV dissociation in VT to be very difficult to differentiate from artifact, as there are always random blips on tachycardia tracings. Cardiology was consulted and the patient underwent coronaryangiogram which showed diffuse severe three-vessel disease. Coronaryangiogram shows diffuse severe three-vessel disease.
The status of the patients chest pain at this time is unknown : EKG 1, 1300: There is sinus tachycardia and artifact of low and high frequency. However, there is also significant tachycardia , with heart rate of 116, and known hypoxia. Acute coronary occlusion and acute pulmonary edema can coexist.
One big chunk of ACS-UA is secondary UA where there is increased demand as in stable angina with tachycardia*. we can witness menacingly deep resting ST depression with absolutely no thrombotic process going on in the coronary. Mind you, even a coronaryangiogram will not bail you out in terms of decision-making and risk prediction.
During observation in the ED the patient had multiple self-terminating runs of Non-Sustained monomorphic Ventricular Tachycardia (NSVT). CT coronaryangiogram showed a hypoplastic RCA and dominant LCx. This patient very likely has some form of idiopathic ventricular tachycardia. No PVCs are seen.
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