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Post cath ECG: Now there are hyperacute T-waves again, and recurrent ST depression in V2 This ECG would normally diagnostic of OMI until proven otherwise No further troponins were measured, but it looks like there is recurrent OMI Next day: A CT CoronaryAngiogram was done (CTCA) CARDIAC MORPHOLOGY AND FUNCTION: 1. IMPRESSION: 1.
A CT Coronaryangiogram was ordered. Here are the results: --Minimally obstructive coronary artery disease. --LAD LAD plaque with 0-25 percent stenosis. The LAD has moderate 40% ostial-proximal LAD stenosis and severe 90% mid LAD stenosis involving first diagonal branch. --The CAD-RADS category 1. --No
History sounds concerning for ACS (could be critical stenosis, triple vessel), but differential also includes dissection, GI bleed, etc. 2 cases of Aortic Stenosis: Diffuse Subendocardial Ischemia on the ECG. We investigated the incidence of an acutely occluded coronary in patients presenting with STE-aVR with multi-lead ST depression.
The coronaryangiogram revealed no critical stenosis, or acute plaque ulceration. Takotsubo should be a diagnosis of exclusion after angiography reveals no obstructive coronary disease, and repeat Echo displays left ventricular recovery. Furthermore, pertinent electrolyte values (e.g. Saini, A., Raymond-Paquin, A.,
More troponin values were measured at the cardiac center: 2327- 267 ng/L 0821- 355 ng/L 1108- 305 ng/L An echocardiogram on day three of the patients admission showed an ejection fraction of 46% with abnormal basal inferior and basal lateral segments, and severe aortic stenosis. What "initiates" the aortic stenosis cascade?
In this case, it is possible that the physicians interpreted the ST depression in anterior leads as subendocardial ischemia of the anterior wall, and the mid LAD stenosis as the culprit of that ischemia. As a result, the diagnostic utility of posterior leads is limited ( See My Comment in the September 21, 2022 post).
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