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Although one may have all kinds of ischemic findings as a result of cardiacarrest (rather than cause of cardiacarrest), this degree of ST elevation and HATW is all but diagnostic of acute proximal LAD occlusion. This rules out subendocardial ischemia and is diagnostic of posterior OMI. V4-5 continue to show STD.
We periodically review this intriguing ECG finding that is best known for its association with hypothermia — but which may also be seen in association with a number of other entities, including acute infarction and cardiacarrest. My Comment addresses a few additional aspects of this phenomenon.
Two recent interventions have proven in randomized trials to improve neurologic survival in cardiacarrest: 1) the combination of the ResQPod and the ResQPump (suction device for compression-decompression CPR -- Lancet 2011 ) and 2) Dual Sequential defibrillation. First — Some thoughts on the post -resuscitation ECG.
Edited by Bracey, Meyers, Grauer, and Smith A 50-something-year-old female with a history of an unknown personality disorder and alcohol use disorder arrived via EMS following cardiacarrest with return of spontaneous circulation. 2022 Jul;27(4):e12939. Epub 2022 Feb 11. Ann Noninvasive Electrocardiol. doi: 10.1111/anec.12939.
His response: “subendocardial ischemia. Smith : It should be noted that, in subendocardial ischemia, in contrast to OMI, absence of wall motion abnormality is common. With the history of Afib, CTA abdomen was ordered to r/o mesenteric ischemia vs ischemic colitis vs small bowel obstruction. Anything more on history?
They had had twice the rate of cardiacarrest and twice the in-hospital mortality[1] In another study of patients diagnosed with STEMI, those on dialysis experienced delayed reperfusion and double the mortality.[2] Problem #1: As I emphasized in My Comment in the December 6, 2022 post — Not all patients with acute MI report chest pain.
The differential is: Posterolateral OMI or subendocardial ischemia The distinction between posterior OMI and subendocardial ischemia can be important and sometimes difficult. Ischemic ST depression includes posterior OMI and subendocardial ischemia. Her prior ECG on file is shown below: What are your next steps?
Blunt Trauma in a Child 40-something male in a head-on Motor Vehicle Collision and Splenic Injury == MY Comment, by K EN G RAUER, MD ( 10/10 /2022 ): == Highly interesting post by Dr. Smith regarding a 30-something male with multiple injuries from a motor vehicle accident. What are the ECG Findings of Cardiac Contusion?
It is apparently fortunate that she had a cardiacarrest; otherwise, her ECG would have been ignored. Then she began complaining of severe dizziness and quickly went into ventricular fibrillation and resuscitation was initiated by hospital staff. She was defibrillated and resuscitated. N Engl J Med [Internet] 2013;368(21):2004–13.
Comment by K EN G RAUER, MD ( 11/28 /2022 ): = Today's case is remarkable for the presence of 2 important ECG findings: i ) Low Voltage ; and , ii ) Electrical Alternans. Alternation in ST segment appearance ( or in the amount of ST elevation or depression ) — is often linked to ischemia. As per Drs.
He developed cardiacarrest shortly after the ECG in Figure-1 was recorded. As reviewed by Almarzuqi et al ( Vasc Health Risk Mgmt 18:397-406, 2022 ) — Potential Causes of Bidirectional VT include: Digitalis toxicity. Acute myocardial ischemia. Cardiac Sarcoidosis. Primary Cardiac Tumors and/or Cardiac Metastasis.
In terms of ischemia, there is both a signal of subendocardial ischemia (STD max in V5-V6 with reciprocal STE in aVR) AND a signal of transmural infarction of the inferior wall with Q wave and STE in lead III with reciprocal STD in I and aVL. He had multiple cardiacarrests with ROSC regained each time.
This was interpreted by the treating clinicians as not showing any evidence of ischemia. The above said — it may prove insightful to take another look at the Wellens' Syndrome case instantly recognized by Dr. Smith in the August 12, 2022 post in Dr. Smith’s ECG Blog. Here is his presenting ECG: ECG 1, t = 0 What do you think?
There is no definite evidence of acute ischemia. (ie, Simply stated — t he patient was having recurrent PMVT without Q Tc prolongation, and without evidence of ongoing transmural ischemia. ( Some residual ischemia in the infarct border might still be present. Both episodes are initiated by an "R-on-T" phenomenon.
mg reduced the risk of cardiovascular death, MI or heart attack, ischemic stroke, or ischemia-driven coronary revascularization by 31% compared with placebo.34 Heart Disease and Stroke Statistics-2022 Update: A Report From the American Heart Association [published correction appears in Circulation. 2022 Sep 6;146(10):e141].
L/min, BP 107/65 mm Hg, HR 71 bpm LVEF 45%, no wall motion abnormality The patient had a complete neurologic recovery, proving once again that the patient is not dead until he/she is warm and dead. == MY Comment by K EN G RAUER, MD ( 2/8/2022 ): == I thought this to be an intriguing case for a number of reasons.
The reason we continue to periodically review cases of Brugada Phenocopy — is that this entity is still overlooked, as it was in today's case ( See My Comment at the bottom of the page in the May 5, 2022 post — and in the November 25, 2022 post of Dr. Smith's ECG Blog — among other cases of Brugada Phenocopy ).
They include myocardial ischemia, acute pericarditis, pulmonary embolism, external compression due to mass over the right ventricular outflow tract region, and metabolic disorders like hyper or hypokalemia and hypercalcemia. 2022 Mar;8(3):386-405. Spontaneous type 1 ECG has the highest number of points at 3.5, 2001 Oct 1;1(1):6-11.
Steve, what do you think of this ECG in this CardiacArrest Patient?" A woman in her 50s with dyspnea and bradycardia A patient with cardiacarrest, ROSC, and right bundle branch block (RBBB). HyperKalemia with CardiacArrest. The April 17, 2022 post ( Leads V1,V2 placed too high ).
Unexplained cardiacarrest or documented VF/polymorphic VT: +3 3. Unexplained sudden cardiac death (3 categories) (+0.5 - +2) 4. Among potential conditions that may produce Brugada Phenocopy are infarction and ischemia. Ischemia or infarction. Cardiacarrest. Clinical History 2.a. Family History 3.a.
Discussion: This patient was very lucky that she had a doctor who understood her initial ECG, advocated for her, performed serial ECGs, and that her serial ECGs happened to have temporary lack of RBBB which reduces the complexity of the ischemia interpretation. Plus recommendations from a 5-member panel on cardiacarrest.
The ECG shows sinus tachycardia, RBBB+LAFB, and signs of anterolateral acute transmural ischemia (most likely due to acute coronary occlusion), with concordant STE in I and aVL, inappropriate STE in V4-6 (though limited a bit by motion, still definite). Plus recommendations from a 5-member panel on cardiacarrest.
Occurrence of “J Waves” in 12-Lead ECG as a Marker of Acute Ischemia and Their Cellular Basis. Can J Cardiol 2022 Kukla P, Jastrezebski M, Praefort W. Occurrence of "J waves" in 12-lead ECG as a marker of acute ischemia and their cellular basis. A prospective evaluation of the electrocardiogr aphic manifestations of hypothermia.
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