Remove 2021 Remove STEMI Remove Tachycardia
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Role of low-density lipoprotein electronegativity and sexual dimorphism in contributing early ventricular tachyarrhythmias following ST-elevation myocardial infarction

Frontiers in Cardiovascular Medicine

Background Early ventricular tachycardia/fibrillation (VT/VF) in patients with ST-elevation myocardial infarction (STEMI) has higher morbidity and mortality. This study examines gender-differentiated risk factors and underlying mechanisms for early onset VT/VF in STEMI. vs. 61.0 ± 13.0 vs. n  = 46, L5: 4.3 ± 9.9%, P  = 0.016).

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ECG Blog #434 — WHY Did this Patient Arrest?

Ken Grauer, MD

Prompt cath is therefore advised if the post-ROSC shows an acute STEMI. The rhythm is regular — at a rate just over 100/minute = sinus tachycardia ( ie, the R-R interval is just under 3 large boxes in duration ). Continuing with assessment of ECG #1 in Figure-2: The rhythm is sinus tachycardia at ~110/minute.

Blog 135
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What are treatment options for this rhythm, when all else fails?

Dr. Smith's ECG Blog

The ECG shows obvious STEMI(+) OMI due to probable proximal LAD occlusion. This progressed to electrical storm , with incessant PolyMorphic Ventricular Tachycardia ( PMVT ) and recurrent episodes of Ventricular Fibrillation ( VFib ). The below ECG was recorded. He required multiple defibrillations within a period of a few hours.

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Hyperkalemia

EMS 12-Lead

The attending crews were concerned for SVT with corresponding ischemic hyperacute T waves (HATW) and subsequently activated STEMI pre-hospital. 2] But there is also Sinus Tachycardia! Then, three minutes later… Crews activated STEMI as she deteriorated into PEA arrest. Closer inspection will show that it is Sinus, after all.

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The Bleeding Heart

EMS 12-Lead

pre-existing, stable atherosclerosis) amidst any state of global duress – to include hypertension, hypoxia, tachycardia, hypotension, sepsis, and GI bleed, for example. STEMI was activated and the patient went to Cath on arrival. There may even be significant overlap between these factors. Journal of Electrocardiology, 61 ; 41-46. [3]

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Infection and DKA, then sudden dyspnea while in the ED

Dr. Smith's ECG Blog

This is ischemic ST depression, and could be due to increasing tachycardia, with a heart rate over 130, but that is unlikely given that the patient is now complaining of crushing chest pain and that there was tachycardia all along. There is widespread ST depression. Figure-1: Comparison of the first 2 ECGs in today's case.

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A man in his 80s with chest pain and ventricular paced rhythm

Dr. Smith's ECG Blog

It was read by the treating physician and the overreading cardiologist as "Paced, no STEMI." As the troponin T was 1521 ng/L (peak troponin T over 1000 ng/L is typical of STEMI) and still rising, no further troponins were measured. Did YOU Notice that the underlying rhythm in Figure-1 appears to be atrial tachycardia?