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Post cath ECG: Now there are hyperacute T-waves again, and recurrent ST depression in V2 This ECG would normally diagnostic of OMI until proven otherwise No further troponins were measured, but it looks like there is recurrent OMI Next day: A CT CoronaryAngiogram was done (CTCA) CARDIAC MORPHOLOGY AND FUNCTION: 1. IMPRESSION: 1.
IMPRESSION: The finding of sinus bradycardia with 1st-degree AV block + marked sinus arrhythmia + the change in PR interval from beat #5-to-beat #6 — suggests a form of vagotonic block ( See My Comment in the October 9, 2020 post in Dr. Smith's ECG Blog ).
CT coronaryangiogram — No obstructive coronary disease. CT coronaryangiogram showed no obstructive coronary disease. But immediate resolution of chest pain once VT was converted — and — the normal CT coronaryangiogram — essentially ruled out acute coronary disease as the cause.
CT coronaryangiogram is excellent , but is rarely available outside of business hours, and hardly ever at night. But this is not the mix-up that occurred in today’s case — because we do not see global negativity ( of P wave, QRS and T wave ) in lead I ( See the February 11, 2020 post). I initially missed that one.
A coronaryangiogram was done that did not show significant coronary artery disease. I have periodically called attention to examples of the Ashman phenomenon as they occur in Dr. Smith's ECG Blog ( See My Comments in the January 5, 2020 post — the June 17, 2020 post — and the March 30, 2023 post , among others ).
Discussion Thus, no further ECGs were recorded and there was no angiogram or stress test or CT coronaryangiogram. IF you missed the KEY Findings on the pre-hospital ECG of todays case Please take another look at My Comment at the bottom of the page of that February 6, 2020 post.
Ct coronaryangiogram showed normal coronary arteries. Smith note: I think CT coronaryangiogram is reasonable with the elevated troponins and symptoms. He was given aspirin and heparin and transferred to the local cardiac center for further evaluation. He was diagnosed with mild AKI which resolved.
A CTCA provides much more anatomical detail and can identify advanced plaque often missed by CT Coronary Artery Calcium Score scans alone. CT Coronary Artery Calcium Score Scan CT Coronary Artery Calcium Score CT CoronaryAngiogram As you can see from the above images, the CTCA provides far more anatomical detail.
It’s judicious, then, to arrange for coronaryangiogram. Coronary occlusion, however, might be present concurrently with subendocardial ischemia on the time-zero ECG, or evolve into such. elevated BP), but rather directly correlated with coronary obstruction and stymied TIMI flow. Does the ECG normalize? 2] Aslanger, E.,
Young people can suffer acute coronary occlusion, whether by typical atherosclerotic plaque rupture, or by coronary anomalies, coronary aneurysms, dissections, spasm, etc. The wall motion abnormalities of Takotsubo cardiomyopathy and LAD OMI can be similar.
Heitner et al found that in 14% of patients with NSTEMI, a blinded interventional cardiologist interpreting coronaryangiograms identified a different culprit artery than CMR ( [link] ). As shown in the mirror-image RED insert Isn't it now obvious that there is acute coronary occlusion causing isolated posterior OMI?
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