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hours T-wave are getting larger again The patient went for an angiogram at about 7 hours after arrival. Angiogram No obstructive epicardial coronary artery disease Cannot exclude non-ACS causes of troponin elevation including coronary vasospasm, stress cardiomyopathy, microvascular disease, etc. IMPRESSION: 1.
IMPRESSION: The finding of sinus bradycardia with 1st-degree AV block + marked sinus arrhythmia + the change in PR interval from beat #5-to-beat #6 — suggests a form of vagotonic block ( See My Comment in the October 9, 2020 post in Dr. Smith's ECG Blog ). This left MINOCA as the most likely cause of this patient's symptoms.
CT coronaryangiogram — No obstructive coronary disease. Subsequent testing supported the presumption of apical cardiomyopathy as the cause of this patient's sustained VT. CT coronaryangiogram showed no obstructive coronary disease. That said — I was not 100% certain about this interpretation.
A coronaryangiogram was done that did not show significant coronary artery disease. The patient was diagnosed with stress cardiomyopathy. Widespread T wave inversions and prolongation of the QT interval is not uncommon in Takotsubo cardiomyopathy. Post ROSC the patient was alert and cooperative.
He was admitted for monitoring, as his risk of a ventricular dysrhythmia as cause of the syncope is high ( very high due to HFrEF and ischemic cardiomyopathy ). Discussion Thus, no further ECGs were recorded and there was no angiogram or stress test or CT coronaryangiogram. The estimated LV ejection fraction is 35%.
Ct coronaryangiogram showed normal coronary arteries. Smith note: I think CT coronaryangiogram is reasonable with the elevated troponins and symptoms. Hypertrophic Cardiomyopathy or Normal ("Variant")? He was given aspirin and heparin and transferred to the local cardiac center for further evaluation.
No family history of sudden cardiac death, cardiomyopathy, premature CAD, or other cardiac issues. Pattern consistent with Takotsubo's cardiomyopathy." Young people can suffer acute coronary occlusion, whether by typical atherosclerotic plaque rupture, or by coronary anomalies, coronary aneurysms, dissections, spasm, etc.
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