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There is a regular narrow complex tachycardia. Thus, it is supraventricular tachycardia. It is important to remember that SVT includes Sinus Tachycardia! Sometimes even Wide Complex Tachycardia is Sinus. See this case in which Lewis leads were necessary to figure this out: Wide Complex Tachycardia.
She had a single chamber ICD/Pacemaker implanted several years prior due to ventricular tachycardia. The ECG was interpreted as showing atrialflutter with 2:1 conduction. Answer : The ECG above shows a regular wide complex tachycardia. The heart rate could be compatible with that of a 2:1 conducted atrialflutter.
There is a regular wide complex tachycardia. A fully upright P-wave is typical atrial activity of atrialflutter as seen in V1. See these example cases of upright P-waves: Case Continued Thus, I was all but certain that this was atrialflutter. If it is flutter, it will reveal the underlying flutter waves.
QUESTIONS: Is this rhythm too fast to be sinus tachycardia? Are flutter waves hidden within the QRS and T waves? However, until such time that we know for certain — I think it best to simply describe what we see: PEARL # 1: Realize that for any tachycardia — there are 6 Parameters that need to be assessed. s in Figure-2 ).
This strip was obtained: Apparent Wide Complex Tachycardia at a rate of 280 What do you think? To me, it was clearly atrialflutter with 1:1 conduction. The rate of 280 is just right for atrialflutter. The waves look like atrialflutter waves, NOT like a wide ventricular complex.
RBBB is no longer seen after conversion to sinus rhythm — which supports our suspicion that the intermittent RBBB conduction seen every-other-beat during the tachycardia ( in Figure-1 ) was rate-related. Note that QRS morphology after conversion to sinus rhythm is very similar to QRS morphology of odd-numbered beats during the tachycardia.
First, we have a narrow-complex, regular tachycardia, with a rate of about 135-140. This narrows our differential for the rhythm down to sinus tachycardia, paroxysmal supraventricular tachycardia (PSVT, or SVT), and atrialflutter. They are flutter waves, and the rhythm is 2:1 atrialflutter.
The rhythm is indeed irregularly irregular, so atrial fibrillation must be considered. There are 5 other rhythms that are irregularly irregular , though atrial fibrillation is by far the most common: 1. Multifocal AtrialTachycardia 2. Sinus with multifocal PACs 3. Sinus with multifocal PVCs 4.
We see a regular tachycardia with a narrow QRS complex and no evidence of OMI or subendocardial ischemia. The differential of a regular narrow QRS tachycardia is sinus tachycardia, SVT, and atrialflutter with regular conduction. There are no P waves preceding the QRS complexes, and no clear flutter waves.
PEARL # 3: AtrialFlutter with 1:1 AV conduction is rare! Since the rate of atrial activity with flutter in adults is most often very close to 300 /minute ( ie, usual range for atrial activity ~250-350/minute ) — AFlutter with 2:1 AV conduction typically results in a regular ventricular rate of ~140-160/minute.
Here is his 12-lead: There is a wide complex tachycardia with a rate of 257, with RBBB and LPFB (right axis deviation) morphology. Read about Fascicular VT here: Idiopathic Ventricular Tachycardias for the EM Physician Case Continued He was completely stable, so adenosine was administered. See Learning point 1 below. Arch Intern Med.
The rhythm is 2:1 atrialflutter. The flutter waves can conceal or mimic ischemic repolarization findings, but here I don't see any obvious findings of OMI or subendocardial ischemia. The rhythm in ECG #1 is not Sinus Tachycardia — because there is no upright P wave in lead II. Here is his triage ECG: What do you think?
Here was his ED ECG: There is sinus tachycardia (rate about 114) with nonspecific ST-T abnormalities. There is a large peaked P-wave in lead II (right atrial enlargement) There is left axis deviation consistent with left anterior fascicular block. See my quick review of atrialtachycardia below) The tachycardia spontaneously resolved.
Grauer at the bottom of the page in the October 26, 2020 post for review of the Cabrera Format ). Figure-1: While at first glance the rhythm in Figure-1 might be mistaken for sinus tachycardia in fact, this is not the rhythm. Instead there is 2:1 atrial activity that is best seen in lead V1 ( See Figure-2 ).
Smith comments : Wide complex tachycardia. The differential diagnosis of WCT is: 1) Sinus tachycardia with "aberrancy" (in this case RBBB and LAFB), but there are no P-waves and the QRS morphology is not typical of simple RBBB/LAFB. Also, if the rate is constant, not wavering up and down, it is highly unlikely to be sinus tachycardia.
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