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EKG with paced complexes shown below shows much narrower QRS complex and echocardiogram showed improved LV systolic function primarily due to improvement in LV dyssynchrony. (J Even with tachycardia and a paced QRS duration of ~0.16 J Am Coll Cardiol. 1 for a rate ~75/min 1. 2 for ~85/min and 1. 3 for ~100/minute ).
Here is her ED ECG: Here is the ED physician's interpretation: IMPRESSION UNCERTAIN REGULAR RHYTHM, wide complex tachycardia, likely p-waves. LEFT BUNDLE BRANCH BLOCK [120+ ms QRS DURATION, 80+ ms Q/S IN V1/V2, 85+ ms R IN I/aVL/V5/V6] Comparison Summary: LBBB and tachycardia are new. This is clearly ventricular tachycardia.
A rapid echocardiogram was performed, revealing an ejection fraction of 20% with thinning of the anterior-apical walls. While the initial impression might not immediately suggest ventricular tachycardia (VT), a closer examination raises suspicion. The initial troponin T level was measured at 30 ng/L. What is the rhythm?
Elevated troponins prompted an echocardiogram — which revealed an apical wall motion abnormality (WMA). NOTE #1: Sinus tachycardia is not usually seen in an uncomplicated acute MI. Patient #1 in today's post did not get expert ECG interpretation. As a result — the heart rate of ~115/minute in ECG #1 is a worrisome finding.
See this case, where a patient with BTWI morphology and dramatic EKG changes within minutes is diagnosed with myocarditis: [link] com/2019/07/what-does-this- ecg-with-significant-st.html EKG 3 also has a saddleback morphology in V2, which is only rarely due to OMI. Still, such dramatic changes cannot be overlooked. It was stented.
Here is his 12-lead: There is a wide complex tachycardia with a rate of 257, with RBBB and LPFB (right axis deviation) morphology. Read about Fascicular VT here: Idiopathic Ventricular Tachycardias for the EM Physician Case Continued He was completely stable, so adenosine was administered. See Learning point 1 below. Learning points 1.
From afar, there is gross tachycardia, cadence irregularities, and narrow QRS complexes that may, or may not, be Sinus in origin; and finally – a cacophony of wide complexes that might very well be ventricular in origin. McLaren : We’ve answered the first question – Sinus Tachycardia with episodic runs of wide QRS (RBBB morphology) and PVC’s.
See this case: what do you think the echocardiogram shows in this case? Am J Med 2019, 132(5):622-630. Now there is a paper published in 2019 that proves the point beyond doubt, though makes it clear that this pattern is associated with very high mortality. American Journal of Medicine 132(5):622-630; May 2019.
The next morning the patient went for his routine echocardiogram, where the operator noticed a dilated aortic root at 5.47 That said — I illustrate HOW this RATIO is arrived at in Figure-2 , which I have adapted from the 3/16/2019 post in Life-In-The-Fast-Lane. Troponins gradually trended down from 0.19
Get an emergent contrast echocardiogram. I learned more about the history: 30-something African American with 5-7days of sharp R-sided shoulder/scapula/chest discomfort, presented with sinus tachycardia. QTc's were 330 ms and 373 ms This is what I texted back: These look like they are a very pronounced case of Benign T-wave Inversion.
Unfortunately there is no echocardiogram accessible because the patient checked himself out of the hospital in order to get back to his home state before it could be completed. C linically — the rhythm we see in the long lead II of ECG #3 behaves similar to MAT, even though there is no tachycardia.
An echocardiogram was done. Kazmi et al have reported on a case in which chest trauma was transiently associated with development of a Brugada-1 ECG pattern ( J Am Coll Cardiol 73 [9-Supp-1], 2019 ). Sinus Tachycardia ( common in any trauma patient. ). Is there also Brugada? Right ventricular prominence.
Here was his ED ECG: There is sinus tachycardia (rate about 114) with nonspecific ST-T abnormalities. Later, he underwent a formal echocardiogram: Very severe left ventricular enlargement (LVED diameter 7.4 An ECG was recorded: This shows a regular narrow complex tachycardia at a rate of about 160. C (99 °F), Resp (!)
We can see enough to make out that the rhythm is sinus tachycardia. Tachycardia is unusual for OMI, unless the patient is in cardiogenic shock (or getting close). A bedside ultrasound should be done to assess volume and other etiologies of tachycardia, but if no cause of type 2 MI is found, the cath lab should be activated NOW.
Otherwise vitals after intubation were only notable for tachycardia. An initial EKG was obtained: Computer read: sinus tachycardia, early acute anterior infarct. A formal echocardiogram was completed the next day and again showed a normal ejection fraction without any focal wall motion abnormalities to suggest CAD.
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