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ACEinhibitors, or potassium-sparing diuretics), are particularly susceptible. A brief review of the pathophysiology of hyperkalemia will help set the stage for the ECG changes inherent therein. Other causes include metabolic acidosis, in which hydrogen ions shift into the cells in exchange for potassium. McCance, K. & Huether, S.
ACEinhibitors, or potassium-sparing diuretics), are particularly susceptible. This ECG is pathognomonic for hyperkalemia. Predisposition to hyperkalemia is complex, but in general, patients with renal disease, or those taking medications that yield potassium retention (e.g. Elsevier-Saunders: Philadelphia, PA. [6]. McCance, K.
associated typical Myocardial Infarction therapies such as statins and ACEinhibitors with significantly decreased 1 year mortality in MINOCA patients, which suggests that they do indeed have a similar pathophysiology to MI patients with obstructive coronary disease. Lindahl et al.
The patient stated he had a long history of well-controlled hypertension for which he was compliant with his ACE-inhibitor. His pain radiated to his shoulders throughout the evening and the night but it did not prompt him to seek medical attention. He was also treated for erectile dysfunction but had not taken any medications recently.
associated typical Myocardial Infarction therapies such as statins and ACEinhibitors with significantly decreased 1 year mortality in MINOCA patients, which suggests that they do indeed have a similar pathophysiology to MI patients with obstructive coronary disease. Lindahl et al.
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