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[link] A 30 year-old woman was brought to the ED with chestpain. She had given birth a week ago, and she had similar chestpain during her labor. She attributed the chestpain to anxiety and stress, saying "I'm just an anxious person." A similar episode had occurred a week earlier during labor.
With more than 500 peer-reviewed publications, the HeartFlow FFR CT Analysis remains unparalleled in precision coronary care, as supported by the ACC/AHA ChestPain Guidelines, to improve treatment plans and outcomes. Neth Heart J 2018. 2021 ACC/AHA ChestPain Guidelines. Arbab-Zadeh, Heart Int 2012. Patel et al.
A 40-something woman had sudden chestpain. Today, they viewed the angiogram and concluded that the thrombus at the mid RCA must have extended proximally from the culprit ruptured plaque, extending proximal to the RV marginal branch and temporarily occluding it. She called 911. But which myocardial walls are affected?
See this post: Classic Evolution of Wellens' T-waves over 26 hours This is one patient, with ECG recorded at time zero after pain resolution, then at time 2 hours, then at time 9 hours. Given that chestpain is resolving at the time ECG #2 was recorded — this supports the concept of dynamic ST-T wave changes in this patient.
The best course is to wait until the anatomy is defined by angio, then if proceeding to PCI, add Cangrelor (an IV P2Y12 inhibitor) I sent the ECG and clinical information of a 90-year old with chestpain to Dr. McLaren. His response: “subendocardial ischemia. Miranda DF, Lobo AS, Walsh B, et al.
They had difficulty describing their symptoms, but complained of severe weakness, nausea, vomiting, headache, and chestpain. They described the chestpain as severe, crushing, and non-radiating. Altogether, this strongly suggests inferolateral OMI, particularly in a patient with acute chestpain.
The CAC scan looks for deposits of calcium in the areas of the coronary arteries as a proxy marker for plaque. It tells you ‘ if ’ there is plaque and how much, as a score called a CAC score. About 1 in 10 patients with a CAC of 0 will have some plaque identified on more in-depth scanning using CTCA 1. Int J Cardiol.
5 Revascularization to improve blood flow to the heart has been shown to reduce mortality in stable chestpain patients. 5 Revascularization to improve blood flow to the heart has been shown to reduce mortality in stable chestpain patients. European Journal of Vascular and Endovascular Surgery, vol. 301–302, [link].
He woke up alert and with chestpain which he also had experienced intermittently over the previous few days. There are multiple possible clinical situations that could account for diffuse subendocardial ischemia that is not due to ACS and plaque rupture. The syncope lasted about 2-3 minutes according to his wife.
A CTCA provides much more anatomical detail and can identify advanced plaque often missed by CT Coronary Artery Calcium Score scans alone. Share ChestPain Symptoms There is no role for CT Calcium Scoring in the setting of someone with chestpain symptoms suspected to be from a narrowed coronary artery.
More than 8 million Americans visit hospital emergency departments experiencing chestpain every year. “In the future, patients with coronary inflammation can be identified by the CaRi-Heart technology and treated by our FDA -approved LODOCO 0.5 mg tablets.”
Whether stenting a narrowed coronary artery improves symptoms such as chestpain (angina) or shortness of breath is a very different question. The cause of angina usually involves inadequate blood flow reaching the heart muscle because of significant narrowing of the artery due to plaque buildup. 2018 Jan 6;391(10115):31-40.
Arteries generally narrow and occlude for one of two reasons: The progressive accumulation of plaque. A plaque ruptures, and a clot forms in the artery, thereby occluding it. There are the ‘garden variety’ heart attacks whereby a plaque ruptures in the coronary artery, called a spontaneous heart attack.
This was diagnosed by IVUS (intravascular ultrasound) as a ruptured plaque. As there was ruptured plaque, this is NOT Prinzmetal's angina. It is just as dangerous, as there is a ruptured plaque with thrombus (which lysed) in the proximal LAD. Values: STE60V3 = 2.0, QRS V2 = 10, RAV4 = 15.5, There was good flow. It was stented.
He had no chestpain, dyspnea, or any other anginal equivalent, and his vital signs were normal. Mechanisms of plaque formation and rupture. Coronary plaque disruption. Just a few weeks ago, I took care of a patient who had ostial RCA OMI (TIMI 0 at cath) and his only complaint was syncope! Virmani, R., & Cammann, V.
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