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ECG in a person with persistent anginal pain for the past several hours showing significant ST segment depression anterolateral leads along with sinus tachycardia. 2016 Sep;68 Suppl 2(Suppl 2):S226-S227. 2016; 6:49-51. ST segment elevation is noted in aVR. Clinical evaluation and X-Ray chest showed features of pulmonary edema.
She was awake, alert, well perfused, with normal mental status and overall unremarkable physical exam except for a regular tachycardia, possible rales at both bases, some mild RUQ abdominal tenderness. Thus, I believe it is a regular, monomorphic, wide complex tachycardia. Or it could simply still be classic VT. What is the Diagnosis?
Otherwise vitals after intubation were only notable for tachycardia. An initial EKG was obtained: Computer read: sinus tachycardia, early acute anterior infarct. Induced Brugada-type electrocardiogram, a sign for imminent malignant arrhythmias. A rectal temperature was obtained which read 107.9 Heart Rhythm, 13(7): 1515-1520. [2]:
Answer : you must treat the patient's underlying condition causing sinus tachycardia, and repeat the ECG at the lower heart rate. Essential Reading : Full text link: AHA/ACCF/HRS Recommendations for the Standardization and Interpretation of the Electrocardiogram, Part IV: The ST Segment, T and U Waves, and the QT Interval (full text link).
If the patient has Abnormal Vital Signs (fever, hypotension, tachycardia, or tachypnea, or hypoxemia), then these are the primary issue to address, as there is ongoing pathology which must be identified. Electrocardiogram-based risk stratification was useful in guiding the use of specialized cardiovascular tests. __ 9) François P.
There is sinus tachycardia and also a large R-wave in aVR. Drug toxicity , especially diphenhydramine , which has sodium channel blocking effects, and also anticholinergic effects which may result in sinus tachycardia, hyperthermia, delirium, and dry skin. 2016) in the presence of spontaneous BrS ECG or drug-induced ECG.
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