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When “spot diagnosing” precordial ST-depression I instinctively evaluate aVR for any corresponding ST-elevation to see if an emerging pattern of broad subendocardial ischemia can be appreciated, in which the ST-depression should be otherwise global and demonstrably maximal in Leads II and V5. ST-elevation, etc.) is present. (A 1] Driver, B.
But it also shows a massive area of total ischemia in the LAD territory: CT shows the infarct The CT is with contrast, which increases density (which looks more white). Angiogram Door to balloon time was 120 minutes (much too long) because of time taken for a CT. No ECG was recorded after pain resolution. 29, 2024 ).
That said there were no clinical symptoms or ECG findings suggestive of ongoing ischemia. CT coronaryangiogram showed a hypoplastic RCA and dominant LCx. In 2016, I doubled the number of night shifts and suddenly was having 10,000 PVCs per day, very distressingly symptomatic. Therefore A different approach is needed.
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