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No ChestPain, but somnolent. The fact that this is syncope makes give it a far lower pretest probability than chestpain, but it was really more than syncope, as the patient actually underwent CPR and had hypotension on arrival of EMS. The American Journal of Emergency Medicine 2015; 33(6):786-790.
A 50-something male who is healthy and active with no previous medical history presented with 5 hours of continuous worrisome chestpain. Chestpain with New LBBB: It helps to actually measure the ST/S ratio A Fascinating Demonstration of ST/S Ratio in LBBB and Resolving LAD Ischemia The cath lab was activated.
== MY Comment by K EN G RAUER, MD ( 9/17/2020 ): == Todays patient is a previously healthy, 60-something year-old woman who presented with chestpain that began at a reception. Smith for developing Modified Smith-Sgarbossa Criteria for assessing ST-T wave changes in chestpain patients with LBBB. See text ). (
Written by Willy Frick A man in his 50s with a history of hypertension, dyslipidemia, type 2 diabetes mellitus, and prior inferior OMI status post DES to his proximal RCA 3 years prior presented to the emergency department at around 3 AM complaining of chestpain onset around 9 PM the evening prior. The following ECG was obtained.
Edits by Meyers and Smith A man in his 70s with PMH of hypertension, hyperlipidemia, type 2 diabetes, CVA, dual-chamber Medtronic pacemaker, presented to the ED for evaluation of acute chestpain. American Heart Journal 170(6):1255-1264; December 2015. Triage ECG: What do you think? This is diagnostic of proximal LAD occlusion.
American Heart Journal 170(6):1255-1264; December 2015. Normal 0 false false false EN-US JA X-NONE Before the case, a few comments: Pendell and I just published a case report of a patient with left bundle branch block who presented with chestpain that then resolved. 0 0 1 41 238 MMRF 1 1 278 14.0
link] A 62 year old man with a history of hypertension, type 2 diabetes mellitus, and carotid artery stenosis called 911 at 9:30 in the morning with complaint of chestpain. He described it as "10/10" intensity, radiating across his chest from right to left. This is written by Willy Frick, an amazing cardiology fellow in St.
Cardiology felt her chestpain to be, most likely, the result of coronary supply-demand mismatch in the context of HCM endothelial remodeling (i.e. A mid-LAD culprit lesion was identified and stented. Below are two examples of this. Type II MI), however decided to pursue coronary angiogram out of an abundance of caution.
Patient 2 : 55 year old with 5 hours of chestpain radiating to the shoulder, with nausea and shortness of breath ECG: sinus bradycardia, normal conduction, normal axis, normal R wave progression, no hypertrophy. This was missed by the treating physician, but the chestpain resolved with aspirin. This was STEMI(-)OMI.
He contacted EMS due to acute onset chestpain and feeling unwell and fatigued. He subsequently developed worsening chestpain. This, in the context of worsening chestpain , is evidence of reocclusion of the infarct-related artery and active OMI in development. The below ECG was recorded. What do you think?
Case A 76 year old man with chronic hypertension but no history of coronary disease or myocardial infarction presented to the ED with chestpain at 2343. It was treated with a drug eluting stent. It is awaiting FDA approval (but approved for 1.5 There is ST elevation in the inferior leads.
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