Remove 2014 Remove Ischemia Remove Myocardial Infarction
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Is this Acute Ischemia? More on LVH.

Dr. Smith's ECG Blog

There may be ischemia present, but it is not evident on the ECG. Journal of Electrocardiology 47 (2014) 655–660. Electrocardiographic Criteria for ST-Elevation Myocardial Infarction in Patients With Left Ventricular Hypertrophy. LVH and the diagnosis of STEMI - how should we apply the current guidelines?

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Is OMI an ECG Diagnosis?

Dr. Smith's ECG Blog

5] Back to the case The patient had serial ECGs over the next hour with no significant change: The first troponin came back at 1,400 ng/L (normal <26 in males and <16 in females), confirming MI – and the patient’s refractory ischemia indicated this was an Occlusion MI. Circulation 2014 2. link] References 1. Amsterdam et al.

STEMI 121
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Diffuse Subendocardial Ischemia on the ECG. Left main? 3-vessel disease? No!

Dr. Smith's ECG Blog

DISCUSSION: The 12-lead EKG EMS initially obtained for this patient showed severe ischemia, with profound "infero-lateral" ST depression and reciprocal ST elevation in lead aVR. The ECG cannot diagnose the etiology of ischemia; it only the presence of ischemia, from whatever etiology.

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Acute OMI or "Benign" Early Repolarization?

Dr. Smith's ECG Blog

The pain will resolve and you will think the ischemia is gone when it is only hidden ! The note also says "slight lateral ST elevations noted, likely early repolarization since unchanged compared to 2014." Smith comment: this troponin alone should be enough data to activate the cath lab, regardless of the ECG. & Griffin, J.

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7 steps to missing posterior Occlusion MI, and how to avoid them

Dr. Smith's ECG Blog

2] Here there is no posterior ST elevation, but the anterior ST depression is also less—so it is dynamic, confirming acute ischemia. The absence of STE in V7-V9 is often due to resolution of ischemia, as seen by resolution of ST depression in V7-V9. V5-V6) of any amplitude, is specific for Occlusion Myocardial Infarction (vs.

STEMI 52
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Chest pain and anterior ST depression. What’s the cause(s)?

Dr. Smith's ECG Blog

WPW, previous Q wave MI, and acute coronary occlusion Depending on the location of the accessory pathway, WPW pattern can mimic ventricular hypertrophy (including RVH or LVH) or myocardial infarction (including anterior, inferior, lateral or posterior MI) [1]. Wolff-Parkinson-White syndrome ‘cured’ by myocardial infarction?

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90 year old with acute chest and epigastric pain, and diffuse ST depression with reciprocal STE in aVR: activate the cath lab?

Dr. Smith's ECG Blog

His response: “subendocardial ischemia. Smith : It should be noted that, in subendocardial ischemia, in contrast to OMI, absence of wall motion abnormality is common. With the history of Afib, CTA abdomen was ordered to r/o mesenteric ischemia vs ischemic colitis vs small bowel obstruction. Anything more on history?