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She was awake, alert, well perfused, with normal mental status and overall unremarkable physical exam except for a regular tachycardia, possible rales at both bases, some mild RUQ abdominal tenderness. Thus, I believe it is a regular, monomorphic, wide complex tachycardia. Or it could simply still be classic VT. What is the Diagnosis?
This strip was obtained: Apparent Wide Complex Tachycardia at a rate of 280 What do you think? Troponins 34>33>43, likely secondary to myocardial injury from tachycardia. Sinus tachycardia does not go this fast. A 60-something ow healthy male had syncope while on treadmill. What do you want to do?
Methods Patients were retrospectively evaluated between January 2012 and June 2020. Objectives To describe a cohort of patients with arrhythmogenic left ventricular cardiomyopathy (ALVC), focusing on the spectrum of the clinical presentations. Significant right ventricular involvement was an exclusion criterion.
There is a run of polymorphic ventricular tachycardia — which given the QT prolongation, qualifies as Torsades de Points ( TdP ). This patient was having recurrent episodes of polymorphic ventricular tachycardia with an underlying long QT interval ( = Torsades des Pointes ). ECG #2 Interpretation of ECG #2: Underlying sinus rhythm.
In that study commonest ECG abnormalites were QTc prolongation followed by brady/tachycardia and then ST segment deviations [3]. 2012 Dec;7(4):290-4. 2012 Dec;7(4):290-4. But the number of persons with lobar hemorrhage in that study was only 17%. Lead electrocardiogram changes after supratentorial intracerebral hemorrhage.
Here was the ECG: There is sinus tachycardia. 109 (20):361-368, 2012 — CLICK HERE ). This was sent by a reader. A previously healthy 53 yo woman was transferred to a receiving hospital in cardiogenic shock. and K was normal. This is "Shark Fin" morphology. C ) ; and , ii ) She tested positive for influenza.
I completely agree with Dr. Nossen that in this patient with new CP and sinus tachycardia with LAHB — that the T waves in each of the inferior leads are hyperacute ( ie, clearly disproportionately "bulky" given size of the QRS in these leads ). — and which other patient(s) need to be seen as soon as possible to figure out what is going on?
NOTE: References I used for my above discussion of BiDirectional VT were — Ali et al ( J Coll Phys and Surgeons Pakistan 23(5):347-349, 2013 ) — and — Femenia, Baranchuk et al ( Indian Pacing Electrophysiol J 12(2):65-68, 2012 ). Contrast today's rhythm with true ventricular bigeminy — that was seen in ECG Blog #343 ).
ANSWER: Treatment of the exceedingly rapid (ie, 1:1 AV conduction ) pattern of AFlutter seen in ECG #1 — resulted in considerable slowing of the rhythm, as seen in ECG #2 : The rhythm in ECG #2 — is a regular tachycardia at a rate just under 150/minute ( ie, about half as fast as the rhythm in ECG #1 ).
Otherwise vitals after intubation were only notable for tachycardia. An initial EKG was obtained: Computer read: sinus tachycardia, early acute anterior infarct. She was ventilated by bag-valve-mask by EMS on arrival and was quickly intubated with etomidate and succinylcholine. A rectal temperature was obtained which read 107.9
We aimed to develop standardized, nationally representative CVD events and selected possible CVD treatment–related complication hospitalization costs for use in cost-effectiveness analyses.METHODS:Nationally representative costs were derived using publicly available inpatient hospital discharge data from the 2012-2018 National Inpatient Sample.
There is sinus tachycardia and also a large R-wave in aVR. Drug toxicity , especially diphenhydramine , which has sodium channel blocking effects, and also anticholinergic effects which may result in sinus tachycardia, hyperthermia, delirium, and dry skin. J Electrocardiology 45 (2012):433-442. Her temperature was 106 degrees.
The relationship between J wave and ventricular tachycardia during Takotsubo cardiomyopathy. Europace 2012 Shinde R, Shinde S, Makhale C, Grant P, Sathe S, Durairaj M, Lokhandwala Y, Di Diego J, Antzelevitch C. Occurrence of “J Waves” in 12-Lead ECG as a Marker of Acute Ischemia and Their Cellular Basis.
There was never ventricular fibrillation (VF) or ventricular tachycardia (VT), no shockable rhythm. Here is a similar case: Collapse, Ventricular Tachycardia, Cardioverted, Comatose on Arrival. Agitation, Confusion, and Unusual Wide Complex Tachycardia. There is sinus tachycardia at ~115/minute.
This was overtaken by a predominance of sympathetic surge ( tachycardia, persistent ST elevation development of electrical "storm" with failure to respond to recurrent defibrillation ). Multidisciplinary critical care management of electrical storm. Journal of the American College of Cardiology , 81 (22), 21892206. link] Mostofsky, E.,
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