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Contemporary data on recurrent AMI and its association with return emergency department (ED) visits for chestpain are needed. Methods This Swedish retrospective cohort study linked patient-level data from six participating hospitals to four national registers to construct the Stockholm Area ChestPain Cohort (SACPC).
A 63 year old man with a history of hypertension, hyperlipidemia, prediabetes, and a family history of CAD developed chestpain, shortness of breath, and diaphoresis after consuming a large meal at noon. He called EMS, who arrived on scene about two hours after the onset of pain to find him hypertensive at 220 systolic.
Two recent interventions have proven in randomized trials to improve neurologic survival in cardiac arrest: 1) the combination of the ResQPod and the ResQPump (suction device for compression-decompression CPR -- Lancet 2011 ) and 2) Dual Sequential defibrillation. Finally, head-up CPR (which was not used here), makes for better resuscitation.
Methods This study included consecutive patients with iSTEMI treated with percutaneous coronary intervention (PCI) between 1 January 2011 and 15 July 2019 at a single, tertiary referral centre.
No chestpain. As noted by Moodie in his manuscript on Adult Congenital Heart Disease ( Tex Heart Inst J: 38(6):705, 2011 ) — there are now more people over the age of 20 with CHD than under that age! Physical exam remarkable for tachypnea ( respiratory rate ~30/minute ) and cyanosis, with clubbing of extremities.
The message is clear — If, in a patient with new chestpain — ST-T wave depression is maximal in leads V2 , V3 and/or V4 — consider acute posterior MI until proven otherwise. In 2011 — Niu et al described the presence of an "N-Wave" — or delayed activation wave of the left ventricular basal region. What is an " N -Wave" ?
This is a 58 year old male with 40 minutes of chestpain of acute onset. He was given aspirin and sublingual nitroglycerine, which improved his pain. Research presented at 2011 SAEM in Boston. The cath lab was activated by the paramedics. On arrival, the following ECG was recorded. 18 (5 Suppl 1):Abstract 425, p.
The patient in today’s case is a previously healthy 40-something male who contacted EMS due to acute onset crushing chestpain. The pain was 10/10 in intensity radiating bilaterally to the shoulders and also to the left arm and neck. Written By Magnus Nossen — with edits by Ken Grauer and Smith. The below ECG was recorded.
If a patient presents with chestpain and a normal heart rate, or with shockable cardiac arrest, then ischemic appearing ST elevation is STEMI until proven otherwise. In this abstract from 2011, we found that 4%(4 of 99) type 2 MI and 38% of type 1 MI had ST Elevation. Clinical Context is everything !
She denied chestpain and denied feeling any palpitations, even during her triage ECG: What do you think? Her symptoms started suddenly about 48 hours ago, but had continued to worsen, including epigastric discomfort, nausea, cough, and dyspnea and lightheadedness on exertion. J Electrocardiol, 42 (2009), pp.
However, according to these diagnostic criteria (JACC 2011; 57(7):802), it is a Bazett corrected QT of less than 330-370, depending on other diagnostic criteria, including 1) h/o cardiac arrest, 2) sudden syncope, 3) family hx of sudden unexplained arrest at age less than 40, 4) family hx of SQTS. short QT syndrome , SQTS).
It was edited by Smith CASE : A 52-year-old male with a past medical history of hypertension and COPD summoned EMS with complaints of chestpain, weakness and nausea. Heart 2011; 97 : 838-843 [link] 14. This was contributed by some folks at Wake Forest: Jason Stopyra, Shannon Mumma, Sean O'Rourke, and Brian Hiestand.
days of chestpain that started as substernal and crushing in nature awakening him from sleep and occasionally traveling to right side of neck. The pain was described as constant, worse with deep inspiration and physical activity, sometimes sharp. He reported 1.5
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