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What are treatment options for this rhythm, when all else fails?

Dr. Smith's ECG Blog

There was no evidence bradycardia leading up to the runs of PMVT ( as tends to occur with Torsades ). If there had been — a temporary atrial pacemaker could have been considered as a way of increasing the heart rate to suppress a bradycardia-dependent arrhythmia ("overdrive pacing").

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QT Correction Formulas Compared to The Rule of Thumb ("Half the RR")

Dr. Smith's ECG Blog

The rule of thumb is less accurate, and the risk is higher because a long QT in the presence of bradycardia ("pause dependent" Torsades) predisposes to Torsades. 6) Use a different rule of thumb for bradycardia : Manually approximate both the QT and the RR interval. 3) At heart rates below 60, far more caution is due.

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Assessment of LV Diastolic Function by Echo in SR and AF

All About Cardiovascular System and Disorders

Use of drugs producing bradycardia like beta blockers in stages III and IV may precipitate low output state. 2011 Dec;19(4):169-73. In stage IV, this restrictive filling pattern remains fixed even during Valsalva maneuver. Initial stages (I to III) are considered reversible with treatment. Stage IV is considered as advanced.

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Tachycardia, fever to 105, and ischemic ST Elevation -- a Bridge too Far

Dr. Smith's ECG Blog

In this abstract from 2011, we found that 4%(4 of 99) type 2 MI and 38% of type 1 MI had ST Elevation. I said I think there is a fixed stenosis in the LAD and the tachycardia and stress caused a type 2 STEMI. link] An angiogram was done: It showed no culprit and no coronary disease, but did show a myocardial bridge in the mid LAD.

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OMI in a pediatric patient? Teenagers do get acute coronary occlusion, so don't automatically dismiss the idea.

Dr. Smith's ECG Blog

A repeat ECG was performed as adult cardiology was asked to evaluate the patient for emerget PCI: Sinus bradycardia with persistent elevation in the inferior leads with reciprocal depression in aVL Patient was taken to cath lab with adult cardiology which revealed normal coronary arteries without evidence of occlusion MI.

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Acute coma, then Sudden PEA arrest in front of paramedics, with STEMI?

Dr. Smith's ECG Blog

Papers I reviewed include ( Poudel et al Cureus 15(6): e40045, 2023 ) ( Chatterjee Neth Heart J 19:31-34, 2011 ) ( Yogendranathan et al BMC Cardiovasc Dis 17(91)- 2017 ) ( Ibrahim and Macdonald Stroke 43(8): 2102-2107, 2012 ) ( Levis Permanente J 21:16-049, 2017 ).

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Electrical instability in a healthy 50 year old. How to manage?

Dr. Smith's ECG Blog

There are 2 main options: Overdrive pacing could be considered and in the right clinical situation, this is often effective for reducing ventricular arrhythmias ( especially in the case of preventing pause induced or bradycardia-induced arrhythmias in association with QTc prolongation ). Try a different kind of antiarrhythmic.