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KEY Point: Look for additional simultaneously-recorded leads = “12 Leads are Better than One!” ( ie, For example with tachycardias — the QRS may look narrow if all you have is 1 or 2 leads — whereas if part of the QRS lies on the baseline in the single lead you are looking at, this might be VT! ). 19:50 — Not appreciating statistical odds! (
22:25 — What if you have a regular SVT ( = narrow-complex tachycardia ) without obvious P waves? ( 2:25 — Dr. Grauer: The 1st Error : Too many clinicians in 2024 are still stuck in the outdated millimeter-based STEMI Paradigm”. ). 19:50 — Not appreciating statistical odds! ( The 4 common causes? — The most commonly overlooked cause? )
T-wave inversions and dynamic ST elevation Tachycardia, hyperthyroid, and ST elevation. Anterior STEMI? A nice Review of EIA by Molis and Molis can be found in Sports Health 2:311-317, 2010. Two cases of ST Elevation with Terminal T-wave Inversion - do either, neither, or both need reperfusion? What is it? Activate the Cath Lab?
This paper by Bischof and Smith compared inferior MI to pericarditis and found that of 154 patients with inferior STEMI, 17% of whom had less than 1 mm of STE in any inferior lead, all 154 had at least 0.25 One looks for sinus tachycardia and diffuse low voltage but many conditions produce these nonspecific findings.
ECG met STEMI criteria and was labeled STEMI by computer interpretation. J waves can also be induced by Occlusion MI (5), STEMI mimics including takotsubo and myocarditis complicated by ventricular arrhythmias (6, 7), and subarachnoid hemorrhage with VF (8). Take home : Not all STEs are STEMIs or OMIs. What do you think?
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