Dr. Paddy Barrett

NOVEMBER 7, 2023

The fundamental characteristic of atherosclerosis is when a cholesterol particle becomes trapped in the artery wall. It is the inflammatory response to this particle retention that causes the formation of atherosclerosis 1. Lipoprotein particles entering the subintimal space causing atherosclerosis. TG = Triglycerides.

Cholesterol 52

Cholesterol Atherosclerosis Stenosis Aortic 52

Dr. S. Venkatesan MD

OCTOBER 2, 2024

So, you want me to teach you the molecular biology of Atherosclerosis ,right ? Atherosclerosis is not a worrisome issue as long as it is contained deep within the vessel wall and doesn’t encroach the lumen. Atherosclerosis. 2009 Feb;202(2):491-7. Does statins increase Thickness of TCFA ? ” Reference 1.van

Plaque 52

Plaque Atherosclerosis Education Cholesterol 52

Dr. Paddy Barrett

AUGUST 22, 2023

Every lipoprotein particle has one APO B protein. ( **Please note the B ) When we measure APO B levels, this is what we are measuring to give an estimate of the number of cholesterol particles and the subsequent risk of cardiovascular disease. APO E is also centrally involved in cholesterol metabolism. Here’s how. What to do?

Cholesterol 52

Cholesterol Dementia Cardiovascular Disease Coronary Artery Disease 52

Stroke Journal

JANUARY 21, 2024

Adipokine, lipid, and immune profiling was conducted. Plaque stability was determined by gold-standard histological classifications. years), the final study analyses included 317 men and 143 women (aged 71.0±9.0

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Plaque Circulation Atherosclerosis Hemorrhage 40